GLUT4 protein is differently modulated during development of obesity in monosodium glutamate-treated mice (Q42524753)

1 November 2017

title

GLUT4 protein is differently modulated during development of obesity in monosodium glutamate-treated mice (English)

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1 November 2017

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4

Maria Tereza Nunes

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1 November 2017

Ubiratan Fabres Machado

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author name string

Paula de Carvalho Papa

series ordinal

1

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1 November 2017

Alessandra Martins Vargas

series ordinal

2

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1 November 2017

José Luciano Tavares da Silva

series ordinal

3

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1 November 2017

language of work or name

English

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publication date

1 September 2002

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1 November 2017

number of pages

12

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based on heuristic

inferred from page(s)

published in

Life Sciences

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1 November 2017

volume

71

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1 November 2017

issue

16

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1 November 2017

page(s)

1917-1928

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The worldwide obesity epidemic

1 November 2017

cites work

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Decreased glucose transporter (GLUT 4) content in insulin-sensitive tissues of obese aurothioglucose- and monosodium glutamate-treated mice

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Reduced BAT function as a mechanism for obesity in the hypophagic, neuropeptide Y deficient monosodium glutamate-treated rat.

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Chronic insulin effects on insulin signalling and GLUT4 endocytosis are reversed by metformin

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PPARgamma induces the insulin-dependent glucose transporter GLUT4 in the absence of C/EBPalpha during the conversion of 3T3 fibroblasts into adipocytes

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Arachidonic acid stimulates glucose uptake in 3T3-L1 adipocytes by increasing GLUT1 and GLUT4 levels at the plasma membrane. Evidence for involvement of lipoxygenase metabolites and peroxisome proliferator-activated receptor gamma.

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0024-3205(02)01948-3

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1 November 2017

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