Contribution of IKCa channels to the control of coronary blood flow. (Q42597971)

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29 January 2020

title

Contribution of IKCa channels to the control of coronary blood flow (English)

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3

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author name string

Michelle M Kurian

series ordinal

1

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29 January 2020

Zachary C Berwick

series ordinal

2

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29 January 2020

publication date

18 April 2011

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Experimental Biology and Medicine

29 January 2020

published in

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29 January 2020

volume

236

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issue

5

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page(s)

621-627

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Matching coronary blood flow to myocardial oxygen consumption.

29 January 2020

cites work

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Role of potassium channels in coronary vasodilation

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Voltage-dependent K+ channels regulate the duration of reactive hyperemia in the canine coronary circulation.

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Role of K(ATP)(+) channels and adenosine in the control of coronary blood flow during exercise.

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Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation

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Regulation of coronary blood flow during exercise

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Bradykinin-induced, endothelium-dependent responses in porcine coronary arteries: involvement of potassium channel activation and epoxyeicosatrienoic acids

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Impaired function of coronary BK(Ca) channels in metabolic syndrome

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Contribution of BK(Ca) channels to local metabolic coronary vasodilation: Effects of metabolic syndrome.

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Characterization of an apamin-sensitive small-conductance Ca(2+)-activated K(+) channel in porcine coronary artery endothelium: relevance to EDHF

21 January 2018

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Characterization of a charybdotoxin-sensitive intermediate conductance Ca2+-activated K+ channel in porcine coronary endothelium: relevance to EDHF.

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Flow-induced dilation of human coronary arterioles: important role of Ca(2+)-activated K(+) channels

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Calcium-activated potassium channels and endothelial dysfunction: therapeutic options?

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Endothelium-dependent relaxation and hyperpolarization evoked by bradykinin in canine coronary arteries: enhancement by exercise-training

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Endothelium-dependent hyperpolarization caused by bradykinin in human coronary arteries

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Ion channel switching and activation in smooth-muscle cells of occlusive vascular diseases

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The intermediate-conductance Ca2+ -activated K+ channel (KCa3.1) in vascular disease.

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Local delivery of the KCa3.1 blocker, TRAM-34, prevents acute angioplasty-induced coronary smooth muscle phenotypic modulation and limits stenosis.

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The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans

21 January 2018

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Delineation of the clotrimazole/TRAM-34 binding site on the intermediate conductance calcium-activated potassium channel, IKCa1

21 January 2018

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Blockade of the intermediate-conductance calcium-activated potassium channel as a new therapeutic strategy for restenosis

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Calcium-activated potassium channels contribute to human coronary microvascular dysfunction after cardioplegic arrest.

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Modulation of coronary autoregulatory responses by endothelium-derived nitric oxide

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Hydrogen peroxide, an endogenous endothelium-derived hyperpolarizing factor, plays an important role in coronary autoregulation in vivo

21 January 2018

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Openers of SKCa and IKCa channels enhance agonist-evoked endothelial nitric oxide synthesis and arteriolar vasodilation

21 January 2018

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Openers of calcium-activated potassium channels and endothelium-dependent hyperpolarizations in the guinea pig carotid artery

21 January 2018

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Leptin receptors are expressed in coronary arteries, and hyperleptinemia causes significant coronary endothelial dysfunction

21 January 2018

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Human coronary arteriolar dilation to arachidonic acid depends on cytochrome P-450 monooxygenase and Ca2+-activated K+ channels.

21 January 2018

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Inhibition of nitric oxide production aggravates myocardial hypoperfusion during exercise in the presence of a coronary artery stenosis.

21 January 2018

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Role of calcium-activated potassium channels with small conductance in bradykinin-induced vasodilation of porcine retinal arterioles.

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