Cell transformation mediated by the Epstein-Barr virus G protein-coupled receptor BILF1 is dependent on constitutive signaling. (Q42663880)

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5 February 2020

title

Cell transformation mediated by the Epstein-Barr virus G protein-coupled receptor BILF1 is dependent on constitutive signaling (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20543866%20AND%20SRC:MED&resulttype=core&format=json

G protein-coupled receptor

5 February 2020

main subject

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5

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5 February 2020

author name string

R Lyngaa

1

1 reference

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5 February 2020

K Nørregaard

2

1 reference

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5 February 2020

M Kristensen

3

1 reference

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5 February 2020

V Kubale

4

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5 February 2020

T N Kledal

6

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5 February 2020

language of work or name

English

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publication date

14 June 2010

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5 February 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20543866%20AND%20SRC:MED&resulttype=core&format=json

5 February 2020

volume

29

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5 February 2020

page(s)

4388-4398

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5 February 2020

issue

31

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https://scigraph.springernature.com/pub.10.1038/onc.2010.173

5 February 2020

exact match

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cites work

Kaposi's sarcoma-associated herpesvirus G protein-coupled receptor constitutively activates NF-kappa B and induces proinflammatory cytokine and chemokine production via a C-terminal signaling determinant

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Human herpesvirus KSHV encodes a constitutively active G-protein-coupled receptor linked to cell proliferation

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G-protein-coupled receptor of Kaposi's sarcoma-associated herpesvirus is a viral oncogene and angiogenesis activator

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The Epstein-Barr virus BILF1 gene encodes a G protein-coupled receptor that inhibits phosphorylation of RNA-dependent protein kinase

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Chemokine sequestration by viral chemoreceptors as a novel viral escape strategy: withdrawal of chemokines from the environment of cytomegalovirus-infected cells

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Kaposi's sarcoma-associated herpesvirus (KSHV) chemokine vMIP-II and human SDF-1alpha inhibit signaling by KSHV G protein-coupled receptor

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Charged residues at the intracellular boundary of transmembrane helices 2 and 3 independently affect constitutive activity of Kaposi's sarcoma-associated herpesvirus G protein-coupled receptor

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Endothelial infection with KSHV genes in vivo reveals that vGPCR initiates Kaposi's sarcomagenesis and can promote the tumorigenic potential of viral latent genes

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Identification and characterization of a G protein-coupled receptor homolog encoded by murine cytomegalovirus.

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inferred from DOI database lookup

7 January 2021

based on heuristic

High constitutive activity of a virus-encoded seven transmembrane receptor in the absence of the conserved DRY motif (Asp-Arg-Tyr) in transmembrane helix 3.

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