Resveratrol attenuates apoptosis of pulmonary microvascular endothelial cells induced by high shear stress and proinflammatory factors (Q42727047)

5 November 2017

Resveratrol attenuates apoptosis of pulmonary microvascular endothelial cells induced by high shear stress and proinflammatory factors (English)

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5 November 2017

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Liang Xia

1

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5 November 2017

Fang Ding

series ordinal

2

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5 November 2017

Jun-Hui Zhu

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3

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5 November 2017

Guo-Sheng Fu

series ordinal

4

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5 November 2017

3 September 2011

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5 November 2017

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5 November 2017

24

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5 November 2017

3

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5 November 2017

127-133

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https://scigraph.springernature.com/pub.10.1007/s13577-011-0031-2

5 November 2017

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Apoptosis of pulmonary microvascular endothelial cells stimulates vascular smooth muscle cell growth

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Cellular and molecular pathobiology of pulmonary arterial hypertension

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Shear stress induces synthetic-to-contractile phenotypic modulation in smooth muscle cells via peroxisome proliferator-activated receptor alpha/delta activations by prostacyclin released by sheared endothelial cells

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Endothelial cell dysfunction and cross talk between endothelium and smooth muscle cells in pulmonary arterial hypertension.

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Resveratrol reduces endothelial progenitor cells senescence through augmentation of telomerase activity by Akt-dependent mechanisms

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Vasoprotective effects of resveratrol and SIRT1: attenuation of cigarette smoke-induced oxidative stress and proinflammatory phenotypic alterations

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Resveratrol increases vascular oxidative stress resistance

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Resveratrol disrupts peroxynitrite-triggered mitochondrial apoptotic pathway: a role for Bcl-2.

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Anti-inflammatory effects of resveratrol: possible role in prevention of age-related cardiovascular disease

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Cells in focus: endothelial cell

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Resveratrol, a polyphenolic compound in red wine, protects against oxidized LDL-induced cytotoxicity in endothelial cells

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Sirtuin activators mimic caloric restriction and delay ageing in metazoans

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Resveratrol protects human endothelium from H(2)O(2)-induced oxidative stress and senescence via SirT1 activation

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Targeting heme oxygenase: therapeutic implications for diseases of the cardiovascular system

21 January 2018

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Pharmacological and clinical aspects of heme oxygenase

21 January 2018

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Peroxisome Proliferator-Activated Receptor Gamma (PPARγ) Expression Is Decreased in Pulmonary Hypertension and Affects Endothelial Cell Growth

21 January 2018

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A new mechanism involving ERK contributes to rosiglitazone inhibition of tumor necrosis factor-alpha and interferon-gamma inflammatory effects in human endothelial cells

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Rosiglitazone reduces glucose-induced oxidative stress mediated by NAD(P)H oxidase via AMPK-dependent mechanism.

21 January 2018

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Initial apoptosis is followed by increased proliferation of apoptosis-resistant endothelial cells

21 January 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21892612

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Exuberant endothelial cell growth and elements of inflammation are present in plexiform lesions of pulmonary hypertension

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21892612

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Resveratrol inhibits macrophage expression of EMMPRIN by activating PPARgamma

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21892612

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1007/S13577-011-0031-2

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Dimensions Publication ID

5 November 2017

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1 reference