Commitment of 3T3-F442A cells to adipocyte differentiation takes place during the first 24-36 h after adipogenic stimulation: TNF-alpha inhibits commitment (Q42803920)

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scientific article published on April 2003

Language	Label	Description	Also known as
English	Commitment of 3T3-F442A cells to adipocyte differentiation takes place during the first 24-36 h after adipogenic stimulation: TNF-alpha inhibits commitment	scientific article published on April 2003

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scholarly article

1 reference

Europe PubMed Central

7 November 2017

Commitment of 3T3-F442A cells to adipocyte differentiation takes place during the first 24-36 h after adipogenic stimulation: TNF-alpha inhibits commitment (English)

1 reference

Europe PubMed Central

7 November 2017

1 reference

based on heuristic

inferred from title

Federico Castro-Munozledo

1

1 reference

Europe PubMed Central

7 November 2017

author name string

Alicia Beltrán-Langarica

2

1 reference

Europe PubMed Central

7 November 2017

Walid Kuri-Harcuch

3

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Europe PubMed Central

7 November 2017

language of work or name

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publication date

1 April 2003

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Europe PubMed Central

7 November 2017

Experimental Cell Research

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Europe PubMed Central

7 November 2017

284

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Europe PubMed Central

7 November 2017

2

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Europe PubMed Central

7 November 2017

163-172

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Europe PubMed Central

7 November 2017

Spontaneous heritable changes leading to increased adipose conversion in 3T3 cells

1 reference

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Adipose cell differentiation in culture

1 reference

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Formation of normally differentiated subcutaneous fat pads by an established preadipose cell line

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A study of the adipose conversion of suspended 3T3 cells by using glycerophosphate dehydrogenase as differentiation marker

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A dual effector theory of growth-hormone action

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Control of the adipogenic differentiation of 3T3-F442A cells by retinoic acid, dexamethasone, and insulin: a topographic analysis

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DNA synthesis and cell division related to adipose differentiation of 3T3 cells

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Conversion of 3T3 fibroblasts into adipose cells: triggering of differentiation by prostaglandin F2alpha and 1-methyl-3-isobutyl xanthine

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Prostacyclin as a potent effector of adipose-cell differentiation

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Differentiation of 3T3-L1 fibroblasts to adipocytes the effect of indomethacin, prostaglandin E1 and cyclic amp on the process of differentiation

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Role of insulin in growth hormone-stimulated 3T3 cell adipogenesis

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Interferon inhibits the conversion of 3T3-L1 mouse fibroblasts into adipocytes

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Inhibition of adipose conversion in 3T3-L2 cells by retinoic acid

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Differentiation of 3T3-F442A cells into adipocytes is inhibited by retinoic acid

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Commitment of adipocyte differentiation in 3T3 cells is inhibited by retinoic acid, and the expression of lipogenic enzymes is modulated through cytoskeleton stabilization

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Type beta transforming growth factor controls the adipogenic differentiation of 3T3 fibroblasts

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Regulation of adipose cell number in man

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Growth hormone and the adipose conversion of 3T3 cells

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Contribution of growth hormone to the adipogenic activity of serum

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Activity of human growth hormone and related polypeptides on the adipose conversion of 3T3 cells

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Differentiation of Ob17 preadipocytes to adipocytes: requirement of adipose conversion factor(s) for fat cell cluster formation

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Adipose conversion of 3T3 cells depends on a serum factor

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Quantitation of adipose conversion and triglycerides by staining intracytoplasmic lipids with Oil red O.

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A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

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Interruption of the adipose conversion of 3T3 cells by biotin deficiency: differentiation without triglyceride accumulation

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Developmentally regulated mRNAs in 3T3-adipocytes: analysis of transcriptional control

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Cachectin/TNF kills or inhibits the differentiation of 3T3-L1 cells according to developmental stage

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TNFalpha disrupts mitotic clonal expansion and regulation of retinoblastoma proteins p130 and p107 during 3T3-L1 adipocyte differentiation

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Transcriptional regulation of gene expression during adipocyte differentiation

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Growth hormone promoted tyrosyl phosphorylation of growth hormone receptors in murine 3T3-F442A fibroblasts and adipocytes

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Growth hormone activates mitogen-activated protein kinase and S6 kinase and promotes intracellular tyrosine phosphorylation in 3T3-F442A preadipocytes

1 reference

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Simultaneous activation of p90rsk and p70s6k S6 kinases by growth hormone in 3T3-F442A preadipocytes

1 reference

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Growth hormone stimulates c-fos gene expression by means of protein kinase C without increasing inositol lipid turnover

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Rapid events in growth hormone action. Induction of c-fos and c-jun transcription in 3T3-F442A preadipocytes

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Cellular and molecular aspects of adipose tissue development

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TNF alpha-mediated inhibition and reversal of adipocyte differentiation is accompanied by suppressed expression of PPARgamma without effects on Pref-1 expression

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Stimulation of adipogenesis in fibroblasts by PPAR gamma 2, a lipid-activated transcription factor

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mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer

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Modulation of adipocyte differentiation by tumor necrosis factor and transforming growth factor beta

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Regulation and expression of retinoblastoma proteins p107 and p130 during 3T3-L1 adipocyte differentiation

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Differentiation-induced gene expression in 3T3-L1 preadipocytes: CCAAT/enhancer binding protein interacts with and activates the promoters of two adipocyte-specific genes.

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Ectopic expression of the CCAAT/enhancer-binding protein alpha promotes the adipogenic program in a variety of mouse fibroblastic cells

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Transdifferentiation of myoblasts by the adipogenic transcription factors PPAR gamma and C/EBP alpha

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Tumor necrosis factor-induced reversal of adipocytic phenotype of 3T3-L1 cells is preceded by a loss of nuclear CCAAT/enhancer binding protein (C/EBP).

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Pref-1, a protein containing EGF-like repeats, inhibits adipocyte differentiation

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TNF-alpha inhibits 3T3-L1 adipocyte differentiation without downregulating the expression of C/EBPbeta and delta

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Tumors secreting human TNF/cachectin induce cachexia in mice

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Purification of cachectin, a lipoprotein lipase-suppressing hormone secreted by endotoxin-induced RAW 264.7 cells

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Selective inhibition of synthesis of enzymes for de novo fatty acid biosynthesis by an endotoxin-induced mediator from exudate cells

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Studies of endotoxin-induced decrease in lipoprotein lipase activity

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Lipoprotein lipase suppression in 3T3-L1 cells by an endotoxin-induced mediator from exudate cells

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10.1016/S0014-4827(02)00036-8

1 reference

Europe PubMed Central

7 November 2017

1 reference

Europe PubMed Central

7 November 2017

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