Deacetylase inhibitors increase muscle cell size by promoting myoblast recruitment and fusion through induction of follistatin (Q42829173)

8 November 2017

title

Deacetylase inhibitors increase muscle cell size by promoting myoblast recruitment and fusion through induction of follistatin (English)

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8 November 2017

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10

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9

Eric P Hoffman

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8 November 2017

Simona Iezzi

1

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8 November 2017

Monica Di Padova

2

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8 November 2017

Carlo Serra

3

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8 November 2017

Giuseppina Caretti

4

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8 November 2017

Cristiano Simone

5

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8 November 2017

Eric Maklan

6

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8 November 2017

Giulia Minetti

7

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8 November 2017

Po Zhao

8

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8 November 2017

Vittorio Sartorelli

11

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8 November 2017

language of work or name

English

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publication date

1 May 2004

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8 November 2017

number of pages

12

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inferred from page(s)

published in

Developmental Cell

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8 November 2017

volume

6

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8 November 2017

issue

5

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8 November 2017

page(s)

673-684

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A dominant-negative inhibitor of CREB reveals that it is a general mediator of stimulus-dependent transcription of c-fos

8 November 2017

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Characterization of a CREB gain-of-function mutant with constitutive transcriptional activity in vivo

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CREB regulates hepatic gluconeogenesis through the coactivator PGC-1

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The myostatin propeptide and the follistatin-related gene are inhibitory binding proteins of myostatin in normal serum

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Regulation of myostatin in vivo by growth and differentiation factor-associated serum protein-1: a novel protein with protease inhibitor and follistatin domains

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IL-4 acts as a myoblast recruitment factor during mammalian muscle growth

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Direct binding of follistatin to a complex of bone-morphogenetic protein and its receptor inhibits ventral and epidermal cell fates in early Xenopus embryo

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Stage-specific modulation of skeletal myogenesis by inhibitors of nuclear deacetylases.

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Regulation of myostatin activity and muscle growth

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The expression of myosin genes in developing skeletal muscle in the mouse embryo

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Histone deacetylase inhibitors as new cancer drugs

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7 January 2021

Multiple defects and perinatal death in mice deficient in follistatin

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Cell Therapy of -Sarcoglycan Null Dystrophic Mice Through Intra-Arterial Delivery of Mesoangioblasts

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7 January 2021

Follistatin: essential role for the N-terminal domain in activin binding and neutralization

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7 January 2021

Deficiency in rhabdomyosarcomas of a factor required for MyoD activity and myogenesis

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Loss of myostatin attenuates severity of muscular dystrophy in mdx mice

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Slug is a novel downstream target of MyoD. Temporal profiling in muscle regeneration

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7 January 2021

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Identifiers

DOI

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8 November 2017

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