Transcriptional mechanisms underlying sensitization of peripheral sensory neurons by granulocyte-/granulocyte-macrophage colony stimulating factors (Q42844654)

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scientific article published on 25 September 2013

Language	Label	Description	Also known as
English	Transcriptional mechanisms underlying sensitization of peripheral sensory neurons by granulocyte-/granulocyte-macrophage colony stimulating factors	scientific article published on 25 September 2013

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scholarly article

1 reference

Europe PubMed Central

8 November 2017

Transcriptional mechanisms underlying sensitization of peripheral sensory neurons by granulocyte-/granulocyte-macrophage colony stimulating factors (English)

1 reference

Europe PubMed Central

8 November 2017

1 reference

based on heuristic

inferred from title

1

1 reference

Europe PubMed Central

8 November 2017

Venkata P Satagopam

3

1 reference

Europe PubMed Central

8 November 2017

author name string

Varun Venkataramani

2

1 reference

Europe PubMed Central

8 November 2017

Pooja Gupta

4

1 reference

Europe PubMed Central

8 November 2017

Reinhard Schneider

5

1 reference

Europe PubMed Central

8 November 2017

Rohini Kuner

6

1 reference

Europe PubMed Central

8 November 2017

publication date

25 September 2013

1 reference

Europe PubMed Central

8 November 2017

1 reference

Europe PubMed Central

8 November 2017

9

1 reference

Europe PubMed Central

8 November 2017

48

1 reference

Europe PubMed Central

8 November 2017

https://scigraph.springernature.com/pub.10.1186/1744-8069-9-48

0 references

Nuclear calcium signaling in spinal neurons drives a genomic program required for persistent inflammatory pain

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Granulocyte-macrophage colony-stimulating factor is a key mediator in experimental osteoarthritis pain and disease development.

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CCL2 and CCL3 are essential mediators of pelvic pain in experimental autoimmune prostatitis

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NOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9

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1 reference

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TRESK channel contribution to nociceptive sensory neurons excitability: modulation by nerve injury.

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Bone cancer pain.

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The IKK-NF-kappaB pathway: a source for novel molecular drug targets in pain therapy?

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Colony-stimulating factors in inflammation and autoimmunity.

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27 May 2018

Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain

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Cytokine regulation of MMP-9 in peripheral glia: implications for pathological processes and pain in injured nerve

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Activation of nuclear factor-kappaB via endogenous tumor necrosis factor alpha regulates survival of axotomized adult sensory neurons.

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Proinflammatory chemokines, such as C-C chemokine ligand 3, desensitize mu-opioid receptors on dorsal root ganglia neurons

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Characterisation of matrix metalloproteinases and the effects of a broad-spectrum inhibitor (BB-1101) in peripheral nerve regeneration

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The JAK/STAT signaling pathway

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Calpain-mediated proteolytic cleavage of the neuronal glycine transporter, GlyT2.

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A decade of caspases

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Matrix metalloproteinases in neuroinflammation

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Organelle-specific initiation of cell death pathways

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Molecular mechanisms of nociception

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Inhibitor kappa B kinase beta dependent cytokine upregulation in nociceptive neurons contributes to nociceptive hypersensitivity after sciatic nerve injury.

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https://pubmed.ncbi.nlm.nih.gov/24067145

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10.1186/1744-8069-9-48

1 reference

Europe PubMed Central

8 November 2017

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1 reference

Europe PubMed Central

8 November 2017

1 reference

Europe PubMed Central

8 November 2017

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