1,2,5,6,9,10-αHexabromocyclododecane (HBCD) impairs thyroid hormone-induced dendrite arborization of Purkinje cells and suppresses thyroid hormone receptor-mediated transcription. (Q42850429)

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scientific article published on March 2011

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English	1,2,5,6,9,10-αHexabromocyclododecane (HBCD) impairs thyroid hormone-induced dendrite arborization of Purkinje cells and suppresses thyroid hormone receptor-mediated transcription.	scientific article published on March 2011

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scholarly article

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15 February 2020

1,2,5,6,9,10-αHexabromocyclododecane (HBCD) impairs thyroid hormone-induced dendrite arborization of Purkinje cells and suppresses thyroid hormone receptor-mediated transcription (English)

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15 February 2020

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Noriyuki Koibuchi

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15 February 2020

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Kingsley Ibhazehiebo

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15 February 2020

Toshiharu Iwasaki

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15 February 2020

Noriaki Shimokawa

3

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1 March 2011

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15 February 2020

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15 February 2020

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15 February 2020

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15 February 2020

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Hexabromocyclododecane challenges scientists and regulators.

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Modulation at a cellular level of the thyroid hormone receptor-mediated gene expression by 1,2,5,6,9,10-hexabromocyclododecane (HBCD), 4,4'-diiodobiphenyl (DIB), and nitrofen (NIP).

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Accumulation and disposition of hexabromocyclododecane (HBCD) in juvenile rainbow trout (Oncorhynchus mykiss).

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Historical review of the significance of the cerebellum and the role of Purkinje cells in motor learning

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Synaptogenesis in the Rat Cerebellum: Effects of Early Hypo- and Hyperthyroidism

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Thyroid hormone-dependent development of mouse cerebellar Purkinje cells in vitro

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ROR alpha augments thyroid hormone receptor-mediated transcriptional activation

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Thyroid hormone response elements differentially modulate the interactions of thyroid hormone receptors with two receptor binding domains in the steroid receptor coactivator-1.

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Polychlorinated biphenyls suppress thyroid hormone receptor-mediated transcription through a novel mechanism

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Identification of the functional domain of thyroid hormone receptor responsible for polychlorinated biphenyl-mediated suppression of its action in vitro

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Thyroid hormone action is disrupted by bisphenol A as an antagonist.

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Polychlorinated biphenyls (PCBs): environmental impact, biochemical and toxic responses, and implications for risk assessment

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Current perspectives on the role of thyroid hormone in growth and development of cerebellum

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Immunofluorescence localization of thyroid hormone receptor protein beta 1 and variant alpha 2 in selected tissues: cerebellar Purkinje cells as a model for beta 1 receptor-mediated developmental effects of thyroid hormone in brain

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Physiological and molecular basis of thyroid hormone action

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The role of thyroid hormones in prenatal and neonatal neurological development--current perspectives

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Identifiers

10.1007/S12311-010-0218-1

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20967578%20AND%20SRC:MED&resulttype=core&format=json

15 February 2020

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20967578%20AND%20SRC:MED&resulttype=core&format=json

15 February 2020

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