Inactivation of Smad4 leads to impaired ocular development and cataract formation (Q42927256)

11 November 2017

title

Inactivation of Smad4 leads to impaired ocular development and cataract formation (English)

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11 November 2017

Ying Liu

1

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11 November 2017

Kirio Kawai

2

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11 November 2017

Shabnam Khashabi

3

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11 November 2017

Chuxia Deng

4

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11 November 2017

Yi-Hsin Liu

5

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11 November 2017

Samuel Yiu

6

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11 November 2017

language of work or name

English

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publication date

22 August 2010

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full work available at URL

11 November 2017

https://api.elsevier.com/content/article/PII:S0006291X10015603?httpAccept=text/xml

1 reference

https://api.crossref.org/works/10.1016/J.BBRC.2010.08.065

https://api.elsevier.com/content/article/PII:S0006291X10015603?httpAccept=text/plain

22 June 2024

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https://api.crossref.org/works/10.1016/J.BBRC.2010.08.065

Biochemical and Biophysical Research Communications

22 June 2024

published in

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11 November 2017

volume

400

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11 November 2017

issue

4

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11 November 2017

page(s)

476-482

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Smad transcription factors

11 November 2017

cites work

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Early eye development in vertebrates

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BMP4 is essential for lens induction in the mouse embryo

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The level of BMP4 signaling is critical for the regulation of distinct T-box gene expression domains and growth along the dorso-ventral axis of the optic cup

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Proper patterning of the optic fissure requires the sequential activity of BMP7 and SHH

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Dual role of the Smad4/DPC4 tumor suppressor in TGFbeta-inducible transcriptional complexes

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Contributions by members of the TGFbeta superfamily to lens development

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Compound developmental eye disorders following inactivation of TGFbeta signaling in neural-crest stem cells

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Reduced programmed cell death in the retina and defects in lens and cornea of Tgfbeta2(-/-) Tgfbeta3(-/-) double-deficient mice

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Differential requirements for Smad4 in TGFbeta-dependent patterning of the early mouse embryo

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Pax6 activity in the lens primordium is required for lens formation and for correct placement of a single retina in the eye

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A requirement for bone morphogenetic protein-7 during development of the mammalian kidney and eye

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BMP7 acts in murine lens placode development

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BMP7 null mutation in mice: developmental defects in skeleton, kidney, and eye.

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Activin/inhibin beta B subunit gene disruption leads to defects in eyelid development and female reproduction.

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Mice homozygous for a null mutation of activin beta B are viable and fertile

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A role for MEK kinase 1 in TGF-beta/activin-induced epithelium movement and embryonic eyelid closure

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MEKK1 transduces activin signals in keratinocytes to induce actin stress fiber formation and migration

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The signaling pathways in tissue morphogenesis: a lesson from mice with eye-open at birth phenotype

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MEK kinase 1 gene disruption alters cell migration and c-Jun NH2-terminal kinase regulation but does not cause a measurable defect in NF-kappa B activation

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JNK initiates a cytokine cascade that causes Pax2 expression and closure of the optic fissure

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ROCK-I regulates closure of the eyelids and ventral body wall by inducing assembly of actomyosin bundles

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FGF-regulated BMP signaling is required for eyelid closure and to specify conjunctival epithelial cell fate

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22 June 2024

Identifiers

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11 November 2017

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