Phosphatidylinositol 3-kinase/Akt induced by erythropoietin renders the erythroid differentiation factor GATA-1 competent for TIMP-1 gene transactivation. (Q42941724)

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scientific article published on September 2005

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English	Phosphatidylinositol 3-kinase/Akt induced by erythropoietin renders the erythroid differentiation factor GATA-1 competent for TIMP-1 gene transactivation.	scientific article published on September 2005

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5 January 2020

Phosphatidylinositol 3-kinase/Akt induced by erythropoietin renders the erythroid differentiation factor GATA-1 competent for TIMP-1 gene transactivation (English)

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5 January 2020

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Paul-Henri Roméo

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Philippe Leboulch

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Philippe Leboulch

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Heather M. Rooke

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Heather M Rooke

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Zahra Kadri

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5 January 2020

Leila Maouche-Chretien

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Stuart H Orkin

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Patrick Mayeux

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Stany Chretien

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1 September 2005

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Molecular and Cellular Biology

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7412-7422

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5 January 2020

GATA-1: friends, brothers, and coworkers.

1 reference

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Gene expression regulation and domain function of hematopoietic GATA factors

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Leukemogenesis caused by incapacitated GATA-1 function

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GATA-1 and NF-Y cooperate to mediate erythroid-specific transcription of Gfi-1B gene

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Erythropoietin induction of tissue inhibitors of metalloproteinase-1 expression and secretion is mediated by mitogen-activated protein kinase and phosphatidylinositol 3-kinase pathways.

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A "knockdown" mutation created by cis-element gene targeting reveals the dependence of erythroid cell maturation on the level of transcription factor GATA-1.

1 reference

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Transcriptional activity of the human tissue inhibitor of metalloproteinases 1 (TIMP-1) gene in fibroblasts involves elements in the promoter, exon 1 and intron 1.

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1 reference

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1 reference

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12 December 2020

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10.1128/MCB.25.17.7412-7422.2005

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5 January 2020

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5 January 2020

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5 January 2020

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