Homeostatic switch in hebbian plasticity and fear learning after sustained loss of Cav1.2 calcium channels. (Q43007346)

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6 February 2020

title

Homeostatic switch in hebbian plasticity and fear learning after sustained loss of Cav1.2 calcium channels (English)

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6 February 2020

6

Sven Moosmang

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6 February 2020

author name string

Nicole Langwieser

1

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6 February 2020

Carl J Christel

2

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6 February 2020

Thomas Kleppisch

3

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6 February 2020

Franz Hofmann

4

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6 February 2020

Carsten T Wotjak

5

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language of work or name

English

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publication date

1 June 2010

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6 February 2020

published in

Journal of Neuroscience

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volume

30

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issue

25

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6 February 2020

page(s)

8367-8375

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Distinct populations of NMDA receptors at subcortical and cortical inputs to principal cells of the lateral amygdala

6 February 2020

cites work

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Functional embryonic cardiomyocytes after disruption of the L-type alpha1C (Cav1.2) calcium channel gene in the mouse.

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NMDA receptors and L-type voltage-gated calcium channels contribute to long-term potentiation and different components of fear memory formation in the lateral amygdala.

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Long-term potentiation in the amygdala: a cellular mechanism of fear learning and memory

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AKAP79/150 anchoring of calcineurin controls neuronal L-type Ca2+ channel activity and nuclear signaling

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A pathway-specific function for different AMPA receptor subunits in amygdala long-term potentiation and fear conditioning

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12 December 2020

Time course and efficiency of protein synthesis inhibition following intracerebral and systemic anisomycin treatment

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L-type voltage-gated calcium channels in conditioned fear: a genetic and pharmacological analysis.

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Selective and protracted effect of nifedipine on fear memory extinction correlates with induced stress response

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12 December 2020

Role of L-type Ca2+ channel isoforms in the extinction of conditioned fear

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Two components of long-term potentiation induced by different patterns of afferent activation

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Nimodipine facilitates associative learning in aging rabbits.

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Intracellular spermine confers rectification on rat calcium-permeable AMPA and kainate receptors

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Block of native Ca(2+)-permeable AMPA receptors in rat brain by intracellular polyamines generates double rectification

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Relative abundance of subunit mRNAs determines gating and Ca2+ permeability of AMPA receptors in principal neurons and interneurons in rat CNS

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Inward rectification of both AMPA and kainate subtype glutamate receptors generated by polyamine-mediated ion channel block

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Contrasting properties of two forms of long-term potentiation in the hippocampus

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Differences in Ca2+ permeability of AMPA-type glutamate receptor channels in neocortical neurons caused by differential GluR-B subunit expression

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Enhanced LTP in mice deficient in the AMPA receptor GluR2.

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12 December 2020

10.1523/JNEUROSCI.4164-08.2010

Identifiers

DOI

1 reference

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6 February 2020

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6 February 2020

PubMed publication ID

20573883

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