Prostacyclin receptor suppresses cardiac fibrosis: role of CREB phosphorylation (Q43093520)

13 November 2017

Prostacyclin receptor suppresses cardiac fibrosis: role of CREB phosphorylation (English)

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13 November 2017

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Caroline J Taylor

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author name string

Elsa C Chan

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13 November 2017

Gregory J Dusting

2

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13 November 2017

Nancy Guo

3

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13 November 2017

Hitesh M Peshavariya

4

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13 November 2017

Rodney Dilley

6

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13 November 2017

Shuh Narumiya

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13 November 2017

Fan Jiang

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13 November 2017

18 April 2010

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Journal of Molecular and Cellular Cardiology

13 November 2017

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13 November 2017

49

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13 November 2017

2

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13 November 2017

176-185

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Mechanisms of cardiac fibrosis in hypertension

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Ventricular remodeling

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Inflammatory mechanisms in myocardial infarction.

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The role of TGF-beta signaling in myocardial infarction and cardiac remodeling

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Involvement of NADPH oxidases in cardiac remodelling and heart failure.

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Transforming growth factor-beta, cell signaling and cardiovascular disorders

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Involvement of the p38 mitogen-activated protein kinase pathway in transforming growth factor-beta-induced gene expression

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Altered transforming growth factor signaling in epithelial cells when ras activation is blocked

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Transforming growth factor-beta promotes pro-fibrotic behavior by serosal fibroblasts via PKC and ERK1/2 mitogen activated protein kinase cell signaling

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Role of p38 MAPK in Transforming Growth Factor β Stimulation of Collagen Production by Scleroderma and Healthy Dermal Fibroblasts

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7 January 2021

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Synergistic cooperation between Sp1 and Smad3/Smad4 mediates transforming growth factor beta1 stimulation of alpha 2(I)-collagen (COL1A2) transcription

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Prostacyclin protects against elevated blood pressure and cardiac fibrosis

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Augmented cardiac hypertrophy in response to pressure overload in mice lacking the prostaglandin I2 receptor

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Prostacyclin derivatives prevent the fibrotic response to TGF-beta by inhibiting the Ras/MEK/ERK pathway

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Involvement of Nox2 NADPH oxidase in adverse cardiac remodeling after myocardial infarction

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Failure of antioxidants to protect against angiotensin II-induced aortic rupture in aged apolipoprotein(E)-deficient mice

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Endogenous relaxin does not affect chronic pressure overload-induced cardiac hypertrophy and fibrosis

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Translation-linked mRNA destabilization accompanying serum-induced Nox4 expression in human endothelial cells.

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Important role of Nox4 type NADPH oxidase in angiogenic responses in human microvascular endothelial cells in vitro

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Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene

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Angiotensin II stimulated expression of transforming growth factor-beta1 in cardiac fibroblasts and myofibroblasts

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Transforming growth factor beta receptor endoglin is expressed in cardiac fibroblasts and modulates profibrogenic actions of angiotensin II

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Interaction between angiotensin II and Smad proteins in fibroblasts in failing heart and in vitro

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Transforming growth factor-beta blockade down-regulates the renin-angiotensin system and modifies cardiac remodeling after myocardial infarction

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cAMP inhibits transforming growth factor-beta-stimulated collagen synthesis via inhibition of extracellular signal-regulated kinase 1/2 and Smad signaling in cardiac fibroblasts

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Angiotensin II directly increases transforming growth factor beta1 and osteopontin and indirectly affects collagen mRNA expression in the human heart.

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TGFbeta, cardiac fibroblasts, and the fibrotic response

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Reduction in Left Ventricular Messenger RNA for Transforming Growth Factor β 1 Attenuates Left Ventricular Fibrosis and Improves Survival Without Lowering Blood Pressure in the Hypertensive TGR(mRen2)27 Rat

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Influence of cytokines and growth factors in ANG II-mediated collagen upregulation by fibroblasts in rats: role of myocytes.

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Transforming growth factor-beta receptor antagonism attenuates myocardial fibrosis in mice with cardiac-restricted overexpression of tumor necrosis factor

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Transforming growth factor beta inhibition increases mortality and left ventricular dilatation after myocardial infarction

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7 January 2021

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13 November 2017

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