Activation of catalase by apelin prevents oxidative stress-linked cardiac hypertrophy (Q43096140)

13 November 2017

title

Activation of catalase by apelin prevents oxidative stress-linked cardiac hypertrophy (English)

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13 November 2017

author

Céline Guilbeau-Frugier

5

object named as

Celine Guilbeau-Frugier

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13 November 2017

Camille Foussal

1

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Olivier Lairez

2

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Denis Calise

3

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Atul Pathak

4

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Philippe Valet

6

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Angelo Parini

7

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Oksana Kunduzova

8

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13 November 2017

language of work or name

English

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publication date

14 April 2010

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13 November 2017

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13 November 2017

volume

584

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13 November 2017

issue

11

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page(s)

2363-2370

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Molecular mechanism of cardiac cellular hypertrophy by mechanical stress

13 November 2017

cites work

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The apelin-APJ system in heart failure: pathophysiologic relevance and therapeutic potential

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Apelin reduces myocardial reperfusion injury independently of PI3K/Akt and P70S6 kinase

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[Pyr1]apelin-13 identified as the predominant apelin isoform in the human heart: vasoactive mechanisms and inotropic action in disease.

21 January 2018

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Oxidative stress-dependent sphingosine kinase-1 inhibition mediates monoamine oxidase A-associated cardiac cell apoptosis

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Apelin protects heart against ischemia/reperfusion injury in rat.

21 January 2018

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21 January 2018

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Down-regulation of catalase and oxidative modification of protein kinase CK2 lead to the failure of apoptosis repressor with caspase recruitment domain to inhibit cardiomyocyte hypertrophy

21 January 2018

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21 January 2018

Identifiers

DOI

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13 November 2017

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