Effect of pressure overload on cardioprotection via PI3K-Akt: comparison of postconditioning, insulin, and pressure unloading (Q43124458)

14 November 2017

Effect of pressure overload on cardioprotection via PI3K-Akt: comparison of postconditioning, insulin, and pressure unloading (English)

1 reference

14 November 2017

Mahmood S Mozaffari

1

1 reference

14 November 2017

Jun Yao Liu

series ordinal

2

1 reference

14 November 2017

Stephen W Schaffer

series ordinal

3

1 reference

14 November 2017

language of work or name

1 reference

18 March 2010

1 reference

Journal of Clinical Hypertension

14 November 2017

1 reference

14 November 2017

23

1 reference

14 November 2017

6

1 reference

14 November 2017

668-674

1 reference

Postconditioning: reduction of reperfusion-induced injury

14 November 2017

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Myocardial protection in reperfusion with postconditioning

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Interaction of cardiovascular risk factors with myocardial ischemia/reperfusion injury, preconditioning, and postconditioning.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Reperfusion injury: does it exist?

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Ischemic postconditioning protects remodeled myocardium via the PI3K-PKB/Akt reperfusion injury salvage kinase pathway.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Mitochondrial K(ATP) channels in preconditioning

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Postconditioning: a form of "modified reperfusion" protects the myocardium by activating the phosphatidylinositol 3-kinase-Akt pathway

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Survival kinases in ischemic preconditioning and postconditioning.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Signalling pathways in ischaemic postconditioning.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Critical nodes in signalling pathways: insights into insulin action

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Signaling pathways in ischemic preconditioning

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

"New"-clear functions of PDK1: beyond a master kinase in the cytosol?

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Ischemia-reperfusion injury and cardioprotection: investigating PTEN, the phosphatase that negatively regulates PI3K, using a congenital model of PTEN haploinsufficiency

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Role of glycogen synthase kinase-3beta in cardioprotection

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Glycogen synthase kinase-3beta phosphorylates Bax and promotes its mitochondrial localization during neuronal apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Mitochondrial permeability transition pore opening as an endpoint to initiate cell death and as a putative target for cardioprotection

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Effect of Hypertension and Hypertension-Glucose Intolerance on Myocardial Ischemic Injury

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Mechanisms underlying afterload-induced exacerbation of myocardial infarct size: role of T-type Ca2+ channel

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Effect of pressure overload on cardioprotection of mitochondrial KATP channels and GSK-3beta: interaction with the MPT pore

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Myocardial protection by insulin at reperfusion requires early administration and is mediated via Akt and p70s6 kinase cell-survival signaling

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Nitric oxide mediates the antiapoptotic effect of insulin in myocardial ischemia-reperfusion: the roles of PI3-kinase, Akt, and endothelial nitric oxide synthase phosphorylation

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Infarct-remodeled myocardium is receptive to protection by isoflurane postconditioning: role of protein kinase B/Akt signaling.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Reversal of hyperglycemic preconditioning by angiotensin II: role of calcium transport

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Insulin withdrawal induces apoptosis via a free radical-mediated mechanism

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Preconditioning and postconditioning: new strategies for cardioprotection.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Early expression of myocardial HIF-1alpha in response to mechanical stresses: regulation by stretch-activated channels and the phosphatidylinositol 3-kinase signaling pathway

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Inducible and cardiac specific PTEN inactivation protects ischemia/reperfusion injury

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Stretch-activated pathways and left ventricular remodeling.

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Beta-adrenergic receptor-stimulated apoptosis in cardiac myocytes is mediated by reactive oxygen species/c-Jun NH2-terminal kinase-dependent activation of the mitochondrial pathway

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Management of hypertension in relation to acute coronary syndromes and revascularisation

1 reference

https://api.crossref.org/works/10.1038%2FAJH.2010.43

7 January 2021

Identifiers

DOI

10.1038/AJH.2010.43

1 reference

14 November 2017

1 reference