SHP2 mediates gp130-dependent cardiomyocyte hypertrophy via negative regulation of skeletal alpha-actin gene (Q43131040)

14 November 2017

SHP2 mediates gp130-dependent cardiomyocyte hypertrophy via negative regulation of skeletal alpha-actin gene (English)

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14 November 2017

Yasushi Fujio

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Issei Komuro

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Issei Komuro

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14 November 2017

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Naoki Mochizuki

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Wataru Shioyama

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14 November 2017

Keiko Yamauchi-Takihara

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object named as

Keiko Yamauchi-Takihara

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Kaori Higuchi

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Tadashi Kuroda

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Satoshi Kunimoto

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14 November 2017

Yoshikazu Nakaoka

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Yoh Arita

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14 November 2017

Kaori Yamamoto

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Keigo Nishida

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Hisao Hirota

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Toshio Hirano

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11 March 2010

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Journal of Molecular and Cellular Cardiology

14 November 2017

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49

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157-164

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Controversies in ventricular remodelling

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Activation of gp130 transduces hypertrophic signal through interaction of scaffolding/docking protein Gab1 with tyrosine phosphatase SHP2 in cardiomyocytes

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Cardiotrophin-1 activates a distinct form of cardiac muscle cell hypertrophy. Assembly of sarcomeric units in series VIA gp130/leukemia inhibitory factor receptor-dependent pathways

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Angiotensin II activates RhoA in cardiac myocytes: a critical role of RhoA in angiotensin II-induced premyofibril formation

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Angiotensin II stimulates alpha-skeletal actin expression in cadiomyocytes in vitro and in vivo in the absence of hypertension

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Interaction of scaffolding adaptor protein Gab1 with tyrosine phosphatase SHP2 negatively regulates IGF-I-dependent myogenic differentiation via the ERK1/2 signaling pathway

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EphA4-mediated Rho activation via Vsm-RhoGEF expressed specifically in vascular smooth muscle cells

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Signaling from G protein-coupled receptors to ERK5/Big MAPK 1 involves Galpha q and Galpha 12/13 families of heterotrimeric G proteins. Evidence for the existence of a novel Ras AND Rho-independent pathway

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Small guanine nucleotide-binding proteins and myocardial hypertrophy

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Regulation of mitogen-activated protein kinases in cardiac myocytes through the small G protein Rac1

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Regulation of Ras and Rho small G proteins by SHP-2.

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Deletion of Ptpn11 (Shp2) in cardiomyocytes causes dilated cardiomyopathy via effects on the extracellular signal-regulated kinase/mitogen-activated protein kinase and RhoA signaling pathways

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beta1 integrin and organized actin filaments facilitate cardiomyocyte-specific RhoA-dependent activation of the skeletal alpha-actin promoter

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Activated MEK5 induces serial assembly of sarcomeres and eccentric cardiac hypertrophy.

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The effects of the selective ROCK inhibitor, Y27632, on ET-1-induced hypertrophic response in neonatal rat cardiac myocytes--possible involvement of Rho/ROCK pathway in cardiac muscle cell hypertrophy.

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Big mitogen-activated protein kinase 1 (BMK1)/extracellular signal regulated kinase 5 (ERK5) is involved in platelet-derived growth factor (PDGF)-induced vascular smooth muscle cell migration

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The neurite outgrowth multiadaptor RhoGAP, NOMA-GAP, regulates neurite extension through SHP2 and Cdc42

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10.1016/J.YJMCC.2010.03.001

7 January 2021

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14 November 2017

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