iNOS contribution to the NMDA-induced excitotoxic lesion in the rat striatum (Q43186148)

15 November 2017

iNOS contribution to the NMDA-induced excitotoxic lesion in the rat striatum (English)

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15 November 2017

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Lecanu L

1

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15 November 2017

Verrecchia C

2

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15 November 2017

Margaill I

3

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15 November 2017

Boulu RG

4

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15 November 2017

Plotkine M

5

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15 November 2017

language of work or name

English

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publication date

1 October 1998

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British Journal of Pharmacology

15 November 2017

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15 November 2017

125

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15 November 2017

3

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15 November 2017

584-590

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Effects of hydrogen peroxide, nitric oxide and antioxidants on NF-κB.

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Aminoguanidine ameliorates and L-arginine worsens brain damage from intraluminal middle cerebral artery occlusion

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Induction of cytosolic NADPH-diaphorase/nitric oxide synthase in reactive microglia/macrophages after quinolinic acid lesions in the rat striatum: an electron and light microscopical study

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Cytokines, endotoxin, and glucocorticoids regulate the expression of inducible nitric oxide synthase in hepatocytes

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Selective potentiation of NMDA-induced neuronal injury following induction of astrocytic iNOS

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Marked induction of calcium-independent nitric oxide synthase activity after focal cerebral ischemia

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Evidence for bidirectional changes in nitric oxide synthase activity in the rat striatum after excitotoxically (quinolinic acid) induced degeneration

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Neuroprotective and deleterious effects of nitric oxide on focal cerebral ischemia-induced neurone death

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The NOS inhibitor, 7-nitroindazole, decreases focal infarct volume but not the response to topical acetylcholine in pial vessels

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Identifiers

DOI

10.1038/SJ.BJP.0702119

1 reference

15 November 2017

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15 November 2017

PubMed publication ID

9806344

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