Immunological dysfunction in HIV-1-infected individuals caused by impairment of adenosine deaminase-induced costimulation of T-cell activation. (Q43196418)

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scientific article published in November 2009

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English	Immunological dysfunction in HIV-1-infected individuals caused by impairment of adenosine deaminase-induced costimulation of T-cell activation.	scientific article published in November 2009

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scholarly article

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16 January 2020

Immunological dysfunction in HIV-1-infected individuals caused by impairment of adenosine deaminase-induced costimulation of T-cell activation (English)

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16 January 2020

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14

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2

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16 January 2020

author name string

José M Martinez-Navio

1

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16 January 2020

Rodrigo Pacheco

3

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16 January 2020

Montserrat Plana

5

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Meritxell Nomdedeu

6

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16 January 2020

Harold Oliva

7

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16 January 2020

Cristina Rovira

8

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16 January 2020

Laia Miralles

9

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16 January 2020

José M Gatell

10

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16 January 2020

Teresa Gallart

11

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16 January 2020

Josefa Mallol

12

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16 January 2020

Carme Lluis

13

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publication date

1 November 2009

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16 January 2020

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16 January 2020

128

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3

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16 January 2020

393-404

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16 January 2020

Role of CD26/dipeptidyl peptidase IV in human T cell activation and function

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Chronic innate immune activation as a cause of HIV-1 immunopathogenesis

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New insights on the perturbations of T cell cycle during HIV infection

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Dendritic cells infected with vpr-positive human immunodeficiency virus type 1 induce CD8+ T-cell apoptosis via upregulation of tumor necrosis factor alpha.

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HIV-1 Vpr causes neuronal apoptosis and in vivo neurodegeneration.

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Role of immune activation in HIV pathogenesis

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Abnormal activation and cytokine spectra in lymph nodes of people chronically infected with HIV-1.

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Cell-mediated immunity and the challenges for vaccine development

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Plasma adenosine deaminase activity among HIV1 Clade C seropositives: relation to CD4 T cell population and antiretroviral therapy.

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Human immunodeficiency virus (HIV) type 1 Vpr induces differential regulation of T cell costimulatory molecules: direct effect of Vpr on T cell activation and immune function

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The Vif and Vpr accessory proteins independently cause HIV-1-induced T cell cytopathicity and cell cycle arrest

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Thymic function in severely immunodeficient HIV type 1-infected patients receiving stable and effective antiretroviral therapy

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The multiple functions of HIV-1 Tat: proliferation versus apoptosis

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Human immunodeficiency virus type 1 Vpr-dependent cell cycle arrest through a mitogen-activated protein kinase signal transduction pathway

1 reference

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CD26, adenosine deaminase, and adenosine receptors mediate costimulatory signals in the immunological synapse.

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Human immunodeficiency virus type 1 Vpr impairs dendritic cell maturation and T-cell activation: implications for viral immune escape

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The role of Vpr in HIV-1 pathogenesis

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Human immunodeficiency virus type 1 gp120 induces abnormal maturation and functional alterations of dendritic cells: a novel mechanism for AIDS pathogenesis.

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Incomplete immune reconstitution after initiation of highly active antiretroviral therapy in human immunodeficiency virus-infected patients with severe CD4+ cell depletion

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Impaired Germinal Center Maturation in Adenosine Deaminase Deficiency

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Nadir CD4+ T-cell count and numbers of CD28+ CD4+ T-cells predict functional responses to immunizations in chronic HIV-1 infection.

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27 August 2018

Human immunodeficiency virus-1 Nef protein interacts with Tat and enhances HIV-1 gene expression

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27 August 2018

Role of HIV-1 Vpr in AIDS pathogenesis: relevance and implications of intravirion, intracellular and free Vpr.

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Immune reconstitution with antiretroviral therapies in chronic HIV-1 infection

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Immune recovery in HIV disease: role of the thymus and T cell expansion in immune reconstitution strategies

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Thymic volume predicts long-term immune reconstitution in HIV-infected children treated with highly active antiretroviral therapy.

1 reference

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CD4 T cell depletion is linked directly to immune activation in the pathogenesis of HIV-1 and HIV-2 but only indirectly to the viral load.

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CD4+ T-lymphocyte nadir and the effect of highly active antiretroviral therapy on phenotypic and functional immune restoration in HIV-1 infection.

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HIV-1 infection impairs cell cycle progression of CD4(+) T cells without affecting early activation responses

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1 reference

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27 August 2018

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1 reference

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Adenosine deaminase isoenzyme levels in patients with human T-cell lymphotropic virus type 1 and human immunodeficiency virus type 1 infections

1 reference

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Selective increase of activation antigens HLA-DR and CD38 on CD4+ CD45RO+ T lymphocytes during HIV-1 infection

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CD8 naive T cell counts decrease progressively in HIV-infected adults

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Expression of ecto-adenosine deaminase and CD26 in human T cells triggered by the TCR-CD3 complex. Possible role of adenosine deaminase as costimulatory molecule

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Selective loss of T cell functions in different stages of HIV infection. Early loss of anti-CD3-induced T cell proliferation followed by decreased anti-CD3-induced cytotoxic T lymphocyte generation in AIDS-related complex and AIDS.

1 reference

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27 August 2018

Low T-cell responsiveness to activation via CD3/TCR is a prognostic marker for acquired immunodeficiency syndrome (AIDS) in human immunodeficiency virus-1 (HIV-1)-infected men.

1 reference

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27 August 2018

Plasma adenosine deaminase2, is a marker for human immunodeficiency virus-1 seroconversion

1 reference

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A role for adenosine deaminase in human monocyte maturation

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Adenosine deaminase activity and acquired immunodeficiency syndrome (AIDS)

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1 December 2018

Elevated adenosine deaminase and purine nucleoside phosphorylase activity in peripheral blood null lymphocytes from patients with acquired immune deficiency syndrome

1 reference

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12 December 2020

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Changes in thymic function in HIV-positive patients treated with highly active antiretroviral therapy and interleukin-2

1 reference

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Nadir CD4+ T Cell Count Predicts Response to Subcutaneous Recombinant Interleukin-2

1 reference

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It is safe to stop antiretroviral therapy in patients with preantiretroviral CD4 cell counts >250 cells/microL

1 reference

https://pubmed.ncbi.nlm.nih.gov/20067539

12 December 2020

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Isolation of lymphocytes, granulocytes and macrophages

1 reference

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T-cell subset alterations and lymphocyte responsiveness to mitogens and antigen during severe primary infection with HIV: a case series of seven consecutive HIV seroconverters

1 reference

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1 reference

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HIV infection of monocytes inhibits the T-lymphocyte proliferative response to recall antigens, via production of eicosanoids

1 reference

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Binding of adenosine deaminase to the lymphocyte surface via CD26

1 reference

https://pubmed.ncbi.nlm.nih.gov/20067539

12 December 2020

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TH1 and TH2 cytokine production by peripheral blood mononuclear cells from HIV-infected patients

1 reference

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Does the HIV-1 manipulate immune network via gp120 immunoglobulin-like domain involving V3 loop?

1 reference

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Unscheduled cyclin B expression and p34 cdc2 activation in T lymphocytes from HIV-infected patients

1 reference

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CD8+ T cells in HIV disease exhibit cytokine receptor perturbation and poor T cell receptor activation but are responsive to gamma-chain cytokine-driven proliferation

1 reference

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Identifiers

10.1111/J.1365-2567.2009.03121.X

1 reference

Europe PubMed Central

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16 January 2020

release_y4w7eln3cndzpcvtnkenrgesei

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https://api.fatcat.wiki/v0/release/y4w7eln3cndzpcvtnkenrgesei

24 November 2022

based on heuristic

mapped directly with Wikidata item

0 references

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20067539%20AND%20SRC:MED&resulttype=core&format=json

16 January 2020

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