Lipopolysaccharide and TNF-alpha modify adenosine A(2A) receptor expression and function in equine monocytes. (Q43200122)

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16 January 2020

title

Lipopolysaccharide and TNF-alpha modify adenosine A(2A) receptor expression and function in equine monocytes (English)

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16 January 2020

1 reference

1

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16 January 2020

Michel L Vandenplas

5

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16 January 2020

author name string

Londa J Berghaus

2

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16 January 2020

James N Moore

3

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16 January 2020

David J Hurley

4

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16 January 2020

Robert Thompson

6

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16 January 2020

Joel Linden

7

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16 January 2020

publication date

11 December 2009

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Veterinary Immunology and Immunopathology

16 January 2020

published in

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16 January 2020

volume

135

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16 January 2020

issue

3-4

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16 January 2020

page(s)

289-295

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A2A adenosine receptor (AR) activation inhibits pro-inflammatory cytokine production by human CD4+ helper T cells and regulates Helicobacter-induced gastritis and bacterial persistence

16 January 2020

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Upregulation of A2A adenosine receptor expression by TNF-alpha in PBMC of patients with CHF: a regulatory mechanism of inflammation

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A2A adenosine receptors and C/EBPbeta are crucially required for IL-10 production by macrophages exposed to Escherichia coli

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Effects of the second-generation synthetic lipid A analogue E5564 on responses to endotoxin in [corrected] equine whole blood and monocytes.

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Inflammatory cytokines regulate function and expression of adenosine A(2A) receptors in human monocytic THP-1 cells.

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Tumor necrosis factor-alpha prevents desensitization of Galphas-coupled receptors by regulating GRK2 association with the plasma membrane

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LPS/TLR4 signal transduction pathway

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IL-1 beta and TNF-alpha regulation of the adenosine receptor (A2A) expression: differential requirement for NF-kappa B binding to the proximal promoter

7 January 2021

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Lipopolysaccharide rapidly modifies adenosine receptor transcripts in murine and human macrophages: role of NF-kappaB in A(2A) adenosine receptor induction

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