Different isoforms of the B-cell mutator activation-induced cytidine deaminase are aberrantly expressed in BCR-ABL1-positive acute lymphoblastic leukemia patients. (Q43275641)

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scientific article published on 17 September 2009

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English	Different isoforms of the B-cell mutator activation-induced cytidine deaminase are aberrantly expressed in BCR-ABL1-positive acute lymphoblastic leukemia patients.	scientific article published on 17 September 2009

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7 February 2020

Different isoforms of the B-cell mutator activation-induced cytidine deaminase are aberrantly expressed in BCR-ABL1-positive acute lymphoblastic leukemia patients (English)

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lymphoblastic leukemia

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Annalisa Lonetti

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Francesca Arruga

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Simona Soverini

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Cristina Papayannidis

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Giuseppe Saglio

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I Iacobucci

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F Messa

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D Cilloni

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F Paoloni

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E Ottaviani

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S Chiaretti

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A Vitale

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F Pane

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P P Piccaluga

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S Paolini

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G Berton

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A Baruzzi

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M Baccarani

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R Foà

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G Martinelli

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17 September 2009

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1

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66-73

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7 February 2020

Philadelphia chromosome-positive acute lymphocytic leukemia

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The biology and therapy of adult acute lymphoblastic leukemia

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Microdeletions are a general feature of adult and adolescent acute lymphoblastic leukemia: Unexpected similarities with pediatric disease

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Activation-induced cytidine deaminase acts as a mutator in BCR-ABL1-transformed acute lymphoblastic leukemia cells

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Specific expression of activation-induced cytidine deaminase (AID), a novel member of the RNA-editing deaminase family in germinal center B cells

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Molecular mechanism of class switch recombination: linkage with somatic hypermutation

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Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme

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Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2)

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The biochemistry of somatic hypermutation

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Molecular mechanisms of antibody somatic hypermutation.

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Expression of the AID protein in normal and neoplastic B cells

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Expression of activation-induced cytidine deaminase in human B-cell non-Hodgkin lymphomas

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Two levels of protection for the B cell genome during somatic hypermutation.

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Activation-induced cytidine deaminase in chronic lymphocytic leukemia B cells: expression as multiple forms in a dynamic, variably sized fraction of the clone.

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Mantle cell lymphoma with t(11;14) and unmutated or mutated VH genes expresses AID and undergoes isotype switch events

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Alternative splicing regulates activation-induced cytidine deaminase (AID): implications for suppression of AID mutagenic activity in normal and malignant B cells

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Long-term outcome of complete cytogenetic responders after imatinib 400 mg in late chronic phase, philadelphia-positive chronic myeloid leukemia: the GIMEMA Working Party on CML.

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A potential graft-versus-leukemia effect after allogeneic hematopoietic stem cell transplantation for patients with Philadelphia chromosome-positive acute lymphoblastic leukemia: results from the French Bone Marrow Transplantation Society.

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Achieving a major molecular response at the time of a complete cytogenetic response (CCgR) predicts a better duration of CCgR in imatinib-treated chronic myeloid leukemia patients.

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Expression of spliced oncogenic Ikaros isoforms in Philadelphia-positive acute lymphoblastic leukemia patients treated with tyrosine kinase inhibitors: implications for a new mechanism of resistance

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Activation-induced cytidine deaminase shuttles between nucleus and cytoplasm like apolipoprotein B mRNA editing catalytic polypeptide 1

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The double-edged sword of activation-induced cytidine deaminase.

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Localization and differential expression of activation-induced cytidine deaminase in the amphibian Xenopus upon antigen stimulation and during early development

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Methylation protects cytidines from AID-mediated deamination

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Genome-wide somatic hypermutation

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H2AX prevents DNA breaks from progressing to chromosome breaks and translocations

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Structure of the DNA deaminase domain of the HIV-1 restriction factor APOBEC3G

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Arf gene loss enhances oncogenicity and limits imatinib response in mouse models of Bcr-Abl-induced acute lymphoblastic leukemia

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Nonparametric Estimation from Incomplete Observations

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BCR–ABL1 lymphoblastic leukaemia is characterized by the deletion of Ikaros

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Estimation of failure probabilities in the presence of competing risks: new representations of old estimators

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Imatinib plus steroids induces complete remissions and prolonged survival in elderly Philadelphia chromosome-positive patients with acute lymphoblastic leukemia without additional chemotherapy: results of the Gruppo Italiano Malattie Ematologiche de

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