Antileukaemic effect of PI3K-mTOR inhibitors in acute myeloid leukaemia-gene expression profiles reveal CDC25B expression as determinate of pharmacological effect (Q43461214)

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scientific article published on 23 October 2013

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English	Antileukaemic effect of PI3K-mTOR inhibitors in acute myeloid leukaemia-gene expression profiles reveal CDC25B expression as determinate of pharmacological effect	scientific article published on 23 October 2013

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scholarly article

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Europe PubMed Central

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23 November 2017

Antileukaemic effect of PI3K-mTOR inhibitors in acute myeloid leukaemia-gene expression profiles reveal CDC25B expression as determinate of pharmacological effect (English)

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Europe PubMed Central

PubMed publication ID

23 November 2017

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based on heuristic

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based on heuristic

inferred from title

Jerome Tamburini

2

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24383842%20AND%20SRC:MED&resulttype=core&format=json

13 June 2020

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Håkon Reikvam

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13 June 2020

Silje Skrede

3

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13 June 2020

Rita Holdhus

4

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13 June 2020

Laury Poulain

5

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13 June 2020

Elisabeth Ersvaer

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13 June 2020

Kimberley J Hatfield

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13 June 2020

Øystein Bruserud

8

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13 June 2020

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publication date

23 October 2013

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23 November 2017

British Journal of Haematology

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13 June 2020

164

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13 June 2020

2

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13 June 2020

200-211

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13 June 2020

CDC25 phosphatases in cancer cells: key players? Good targets?

1 reference

https://api.crossref.org/works/10.1111%2FBJH.12611

21 January 2018

CDC25B involvement in the centrosome duplication cycle and in microtubule nucleation

1 reference

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Therapeutic targeting of NF-κB in myelodysplastic syndromes and acute myeloid leukaemia – the biological heterogeneity

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CDC25B mediates rapamycin-induced oncogenic responses in cancer cells.

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Cysteine 81 is critical for the interaction of S100A4 and myosin-IIA

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CDC25 phosphatases as potential human oncogenes

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Cdc25 phosphatases and cancer

1 reference

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Identification of a potent and selective pharmacophore for Cdc25 dual specificity phosphatase inhibitors

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Acute myeloid leukemia: the challenge of capturing disease variety

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Combined inhibition of PI3K and mTOR exerts synergistic antiproliferative effect, but diminishes differentiative properties of rapamycin in acute myeloid leukemia cells.

1 reference

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The FOXM1 transcriptional factor promotes the proliferation of leukemia cells through modulation of cell cycle progression in acute myeloid leukemia.

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Transcriptional repression of Cdc25B by IER5 inhibits the proliferation of leukemic progenitor cells through NF-YB and p300 in acute myeloid leukemia

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Amino acids mediate mTOR/raptor signaling through activation of class 3 phosphatidylinositol 3OH-kinase.

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Role of the PI3K/AKT and mTOR signaling pathways in acute myeloid leukemia.

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Prognostic relevance of integrated genetic profiling in acute myeloid leukemia

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A phase I study of the mammalian target of rapamycin inhibitor sirolimus and MEC chemotherapy in relapsed and refractory acute myelogenous leukemia.

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Antileukemic activity of rapamycin in acute myeloid leukemia.

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21 January 2018

Expression profile of heat shock proteins in acute myeloid leukaemia patients reveals a distinct signature strongly associated with FLT3 mutation status--consequences and potentials for pharmacological intervention.

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21 January 2018

Pharmacological targeting of the PI3K/mTOR pathway alters the release of angioregulatory mediators both from primary human acute myeloid leukemia cells and their neighboring stromal cells

1 reference

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21 January 2018

Differences in proliferative capacity of primary human acute myelogenous leukaemia cells are associated with altered gene expression profiles and can be used for subclassification of patients

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A phase 2 clinical trial of deforolimus (AP23573, MK-8669), a novel mammalian target of rapamycin inhibitor, in patients with relapsed or refractory hematologic malignancies

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Mutations and treatment outcome in cytogenetically normal acute myeloid leukemia

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Protein synthesis is resistant to rapamycin and constitutes a promising therapeutic target in acute myeloid leukemia.

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βTrCP-dependent degradation of CDC25B phosphatase at the metaphase-anaphase transition is a pre-requisite for correct mitotic exit.

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Significance analysis of microarrays applied to the ionizing radiation response

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The use of housekeeping genes for real-time PCR-based quantification of fusion gene transcripts in acute myeloid leukemia

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Discovery of New Inhibitors of Cdc25B Dual Specificity Phosphatases by Structure-Based Virtual Screening

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