Arachidonic acid causes cell death through the mitochondrial permeability transition. Implications for tumor necrosis factor-alpha aopototic signaling (Q43514127)

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English	Arachidonic acid causes cell death through the mitochondrial permeability transition. Implications for tumor necrosis factor-alpha aopototic signaling	scientific article

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scholarly article

1 reference

Europe PubMed Central

24 November 2017

Arachidonic acid causes cell death through the mitochondrial permeability transition. Implications for tumor necrosis factor-alpha aopototic signaling (English)

1 reference

Europe PubMed Central

24 November 2017

1 reference

based on heuristic

inferred from title

1

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Europe PubMed Central

24 November 2017

5

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Europe PubMed Central

24 November 2017

Valeria Petronilli

object named as

V Petronilli

3

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author name string

D Penzo

2

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Europe PubMed Central

24 November 2017

F Pagano

4

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Europe PubMed Central

24 November 2017

language of work or name

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publication date

27 December 2000

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Europe PubMed Central

24 November 2017

number of pages

6

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Journal of Biological Chemistry

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Europe PubMed Central

24 November 2017

276

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Europe PubMed Central

24 November 2017

15

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Europe PubMed Central

24 November 2017

12035-12040

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Europe PubMed Central

24 November 2017

On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Bcl-2 inhibits the mitochondrial release of an apoptogenic protease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Apoptosis inducing factor (AIF): a phylogenetically old, caspase-independent effector of cell death

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Tumor necrosis factor receptor and Fas signaling mechanisms.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Requirement for GD3 ganglioside in CD95- and ceramide-induced apoptosis.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Commitment to apoptosis by GD3 ganglioside depends on opening of the mitochondrial permeability transition pore.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

The cytotoxicity of tumor necrosis factor depends on induction of the mitochondrial permeability transition

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Ceramide induces hepatocyte cell death through disruption of mitochondrial function in the rat.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Ordering of ceramide formation, caspase activation, and mitochondrial changes during CD95- and DNA damage-induced apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Apoptosis driven by IP(3)-linked mitochondrial calcium signals

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Involvement of caspase-dependent activation of cytosolic phospholipase A2 in tumor necrosis factor-induced apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Phospholipase A2 is necessary for tumor necrosis factor alpha-induced ceramide generation in L929 cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

On the effects of paraquat on isolated mitochondria. Evidence that paraquat causes opening of the cyclosporin A-sensitive permeability transition pore synergistically with nitric oxide.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Rhodamine 123 as a probe of transmembrane potential in isolated rat-liver mitochondria: spectral and metabolic properties

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Effect of fatty acids on energy coupling processes in mitochondria

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Effects of aristolochic acid on phospholipase A2 activity and arachidonate metabolism of human neutrophils

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Fatty acid-promoted mitochondrial permeability transition by membrane depolarization and binding to the ADP/ATP carrier.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Fatty acid-induced uncoupling of oxidative phosphorylation is partly due to opening of the mitochondrial permeability transition pore.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Modulation of the mitochondrial permeability transition pore by pyridine nucleotides and dithiol oxidation at two separate sites.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Inhibition of the mitochondrial permeability transition by cyclosporin A during long time frame experiments: relationship between pore opening and the activity of mitochondrial phospholipases.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Mitochondria and cell death. Mechanistic aspects and methodological issues

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

The mitochondrial permeability transition is required for tumor necrosis factor alpha-mediated apoptosis and cytochrome c release

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Cytochrome c-dependent activation of caspase-3 by tumor necrosis factor requires induction of the mitochondrial permeability transition

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Physiological regulation of the transport activity in the uncoupling proteins UCP1 and UCP2.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Fatty acid uncoupling of oxidative phosphorylation in rat liver mitochondria.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

On the nature of the uncoupling effect of fatty acids

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

The ATP/ADP-antiporter is involved in the uncoupling effect of fatty acids on mitochondria.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Effects of arachidonic acid on respiratory activities in isolated brain mitochondria

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Nonsteroidal anti-inflammatory drugs and cancer prevention

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Mechanisms underlying nonsteroidal antiinflammatory drug-mediated apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Intracellular unesterified arachidonic acid signals apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Brain free fatty acids, edema, and mortality in gerbils subjected to transient, bilateral ischemia, and effect of barbiturate anesthesia.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

A combined light and electron microscope study of early anoxic-ischaemic cell change in rat brain

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Amelioration by cyclosporin A of brain damage following 5 or 10 min of ischemia in rats subjected to preischemic hyperglycemia.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Direct Effect of Ceramide on the Mitochondrial Electron Transport Chain Leads to Generation of Reactive Oxygen Species

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Regulation of the permeability transition pore, a voltage-dependent mitochondrial channel inhibited by cyclosporin A

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

The Mitochondrial Permeability Transition Pore is Modulated by Oxidative Agents Through Both Pyridine Nucleotides and Glutathione at Two Separate Sites

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Chemical Modification of Arginines by 2,3-Butanedione and Phenylglyoxal Causes Closure of the Mitochondrial Permeability Transition Pore

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M010603200

21 January 2018

Identifiers

10.1074/JBC.M010603200

1 reference

Europe PubMed Central

24 November 2017

1 reference

Europe PubMed Central

24 November 2017

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