Distribution of tripeptidyl peptidase I in human tissues under normal and pathological conditions (Q43540060)

24 November 2017

Distribution of tripeptidyl peptidase I in human tissues under normal and pathological conditions (English)

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24 November 2017

Kida E

1

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24 November 2017

Golabek AA

2

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24 November 2017

Walus M

3

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24 November 2017

Wujek P

4

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24 November 2017

Kaczmarski W

5

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24 November 2017

Wisniewski KE

6

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24 November 2017

1 March 2001

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Journal of Neuropathology & Experimental Neurology

24 November 2017

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24 November 2017

60

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24 November 2017

3

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24 November 2017

280-292

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Tripeptidyl peptidases: enzymes that count

24 November 2017

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https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Active site of tripeptidyl peptidase II from human erythrocytes is of the subtilisin type

21 January 2018

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https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Purification and characterisation of a tripeptidyl aminopeptidase I from rat spleen.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Association of mutations in a lysosomal protein with classical late-infantile neuronal ceroid lipofuscinosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Mutational analysis of the defective protease in classic late-infantile neuronal ceroid lipofuscinosis, a neurodegenerative lysosomal storage disorder

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Tripeptidyl-peptidase I is apparently the CLN2 protein absent in classical late-infantile neuronal ceroid lipofuscinosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Variability in the clinical and pathological findings in the neuronal ceroid lipofuscinoses: Review of data and observations

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Reevaluation of neuronal ceroid lipofuscinoses: atypical juvenile onset may be the result of CLN2 mutations.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Biochemical characterization of a lysosomal protease deficient in classical late infantile neuronal ceroid lipofuscinosis (LINCL) and development of an enzyme-based assay for diagnosis and exclusion of LINCL in human specimens and animal models

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Increased brain lysosomal pepstatin-insensitive proteinase activity in patients with neurodegenerative diseases.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Classical late infantile neuronal ceroid lipofuscinosis fibroblasts are deficient in lysosomal tripeptidyl peptidase I

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

The expression of late infantile neuronal ceroid lipofuscinosis (CLN2) gene product in human brains.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

A lysosomal proteinase, the late infantile neuronal ceroid lipofuscinosis gene (CLN2) product, is essential for degradation of a hydrophobic protein, the subunit c of ATP synthase.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Purification and characterization of a tripeptidyl peptidase I from human osteoclastomas: evidence for its role in bone resorption

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Structural organization and sequence of CLN2, the defective gene in classical late infantile neuronal ceroid lipofuscinosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Purification and characterization of bovine brain lysosomal pepstatin-insensitive proteinase, the gene product deficient in the human late-infantile neuronal ceroid lipofuscinosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Expression studies of CLN3 protein (battenin) in fusion with the green fluorescent protein in mammalian cells in vitro

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Infantile type of so-called neuronal ceroid-lipofuscinosis. Histological and electron microscopic studies

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Mechanisms of angiogenesis in the brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Structure of the human lysosomal acid phosphatase gene

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Structure and sequence of the human alpha-L-iduronidase gene

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Multi-level regulation of lysosomal gene expression in lymphocytes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Estradiol Down-Regulates the Mannose-6-Phosphate/Insulin-Like Growth Factor-II Receptor Gene and Induces Cathepsin-D in Breast Cancer Cells: A Receptor Saturation Mechanism to Increase the Secretion of Lysosomal Proenzymes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Lysosomal biogenesis in lysosomal storage disorders

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Lysosomal hydrolases of different classes are abnormally distributed in brains of patients with Alzheimer disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

Topographic heterogeneity of amyloid B-protein epitopes in brains with various forms of neuronal ceroid lipofuscinoses suggesting defective processing of amyloid precursor protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F60.3.280

21 January 2018

Identifiers

DOI

10.1093/JNEN/60.3.280

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24 November 2017

1 reference