Post-ischemic PKC inhibition impairs myocardial calcium handling and increases contractile protein calcium sensitivity. (Q43634903)

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8 November 2019

title

Post-ischemic PKC inhibition impairs myocardial calcium handling and increases contractile protein calcium sensitivity (English)

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8 November 2019

5

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8 November 2019

author name string

C Stamm

1

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8 November 2019

I Friehs

2

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8 November 2019

D B Cowan

3

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8 November 2019

H Cao-Danh

4

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8 November 2019

M Munakata

6

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8 November 2019

F X McGowan

7

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8 November 2019

P J del Nido

8

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8 November 2019

publication date

1 July 2001

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8 November 2019

published in

Cardiovascular Research

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8 November 2019

volume

51

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8 November 2019

issue

1

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8 November 2019

page(s)

108-121

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Myocardial stunning and hibernation. The physiology behind the colloquialisms

8 November 2019

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Tonic regulation of excitation-contraction coupling by basal protein kinase C activity in isolated cardiac myocytes

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Differential regulation of cardiac actomyosin S-1 MgATPase by protein kinase C isozyme-specific phosphorylation of specific sites in cardiac troponin I and its phosphorylation site mutants

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Long-term expression of protein kinase C in adult mouse hearts improves postischemic recovery

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Relation between steady-state force and intracellular [Ca2+] in intact human myocardium. Index of myofibrillar responsiveness to Ca2+.

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A protein kinase C translocation inhibitor as an isozyme-selective antagonist of cardiac function

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Ischemic Preconditioning Induces Selective Translocation of Protein Kinase C Isoforms and in the Heart of Conscious Rabbits Without Subcellular Redistribution of Total Protein Kinase C Activity

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Protein kinase C-epsilon is responsible for the protection of preconditioning in rabbit cardiomyocytes

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Activation of epsilon protein kinase C correlates with a cardioprotective effect of regular ethanol consumption

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Phosphorylation by protein kinase C and the responsiveness of Mg(2+)-ATPase to Ca2+ of myofibrils isolated from stunned and non-stunned porcine myocardium.

21 January 2018

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Troponin I degradation and covalent complex formation accompanies myocardial ischemia/reperfusion injury

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Relationship between intracellular calcium and contractile force in stunned myocardium. Direct evidence for decreased myofilament Ca2+ responsiveness and altered diastolic function in intact ventricular muscle

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Protein kinase C activation in the heart: effects on calcium and contractile proteins

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Protein kinase C phosphorylation of cardiac troponin I and troponin T inhibits Ca(2+)-stimulated MgATPase activity in reconstituted actomyosin and isolated myofibrils, and decreases actin-myosin interactions

7 January 2021

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Protein kinase C activation before cardioplegic arrest: beneficial effects on myocyte contractility

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Cardiac preconditioning with calcium: clinically accessible myocardial protection

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Protein kinase C activates ATP-sensitive K+ current in human and rabbit ventricular myocytes.

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Protein kinase C translocation and PKC-dependent protein phosphorylation during myocardial ischemia

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Two distinct mechanisms mediate a differential regulation of protein kinase C isozymes in acute and prolonged myocardial ischemia

7 January 2021

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Role of protein kinase C in mitochondrial KATP channel-mediated protection against Ca2+ overload injury in rat myocardium

7 January 2021

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Phorbol ester and dioctanoylglycerol stimulate membrane association of protein kinase C and have a negative inotropic effect mediated by changes in cytosolic Ca2+ in adult rat cardiac myocytes

7 January 2021

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Effect of protein kinase C activation on sarcoplasmic reticulum function and apparent myofibrillar Ca2+ sensitivity in intact and skinned muscles from normal and diseased human myocardium

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Anti-ischemic effect of a novel cardioprotective agent, JTV519, is mediated through specific activation of delta-isoform of protein kinase C in rat ventricular myocardium

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10.1016/S0008-6363(01)00249-8

7 January 2021

Identifiers

DOI

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8 November 2019

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