Effect of Hydroxysafflor yellow A on human umbilical vein endothelial cells under hypoxia (Q43673013)

25 November 2017

title

Effect of Hydroxysafflor yellow A on human umbilical vein endothelial cells under hypoxia (English)

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25 November 2017

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Deng Bo Ji

1

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25 November 2017

Li Yun Zhang

2

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25 November 2017

Chang Ling Li

3

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25 November 2017

Jia Ye

4

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25 November 2017

Hai Bo Zhu

5

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25 November 2017

publication date

28 November 2008

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25 November 2017

published in

Vascular Pharmacology

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25 November 2017

volume

50

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25 November 2017

issue

3-4

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25 November 2017

page(s)

137-145

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Endothelium-derived relaxing factor from cultured human endothelial cells inhibits aggregation of human platelets.

25 November 2017

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Protective effects of hydroxysafflor yellow A on acute and chronic congestive cardiac failure mediated by reducing ET-1, NOS and oxidative stress in rats

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Hypoxia-induced endothelial apoptosis through nuclear factor-kappaB (NF-kappaB)-mediated bcl-2 suppression: in vivo evidence of the importance of NF-kappaB in endothelial cell regulation

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Hydroxysafflor yellow A enhances survival of vascular endothelial cells under hypoxia via upregulation of the HIF-1 alpha-VEGF pathway and regulation of Bcl-2/Bax

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Nitric oxide: an endogenous modulator of leukocyte adhesion

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Glucosaminylmuramyl dipeptide (GMDP) modulates endothelial cell activities in vitro but has no effect on angiogenesis in vivo

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Hypoxia inhibits expression of eNOS via transcriptional and posttranscriptional mechanisms

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Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

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Different signaling pathways induce apoptosis in endothelial cells and cardiac myocytes during ischemia/reperfusion injury

7 January 2021

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Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications

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c-Jun triggers apoptosis in human vascular endothelial cells

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7 January 2021

Identifiers

DOI

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25 November 2017

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