Acute actions of tumor necrosis factor-alpha on intracellular Ca(2+) and K(+) currents in human microglia (Q43678444)

25 November 2017

Acute actions of tumor necrosis factor-alpha on intracellular Ca(2+) and K(+) currents in human microglia (English)

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25 November 2017

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McLarnon JG

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25 November 2017

Franciosi S

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25 November 2017

Wang X

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25 November 2017

Bae JH

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25 November 2017

Choi HB

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25 November 2017

Kim SU

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25 November 2017

language of work or name

English

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publication date

1 January 2001

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25 November 2017

10

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inferred from page(s)

published in

Neuroscience

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25 November 2017

104

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25 November 2017

4

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25 November 2017

1175-1184

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Bacterial endotoxin induces [Ca2+]i transients and changes the organization of actin in microglia

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Prostaglandin E2 and 4-aminopyridine prevent the lipopolysaccharide-induced outwardly rectifying potassium current and interleukin-1beta production in cultured rat microglia

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Human immunodeficiency virus and the brain

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Acute application of interleukin-1beta induces Ca(2+) responses in human microglia

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Tumor necrosis factor identified in multiple sclerosis brain

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Complement factor C5a and epidermal growth factor trigger the activation of outward potassium currents in cultured murine microglia.

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Modulation of potassium currents in cultured murine microglial cells by receptor activation and intracellular pathways

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TNF alpha induces a protein kinase C-dependent reduction in astroglial K+ conductance.

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Tumour necrosis factor-alpha increases intracellular Ca2+ and induces a depolarization in cultured astroglial cells.

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G protein control of potassium channel activity in a mast cell line

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G-protein activators induce a potassium conductance in murine macrophages

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Effects of ATP and elevated K+ on K+ currents and intracellular Ca2+ in human microglia

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Tumor necrosis factor alpha-induced phosphorylation of I kappa B alpha is a signal for its degradation but not dissociation from NF-kappa B

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Complement 5a controls motility of murine microglial cells in vitro via activation of an inhibitory G-protein and the rearrangement of the actin cytoskeleton

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Inflammatory stimuli induce a new K+ outward current in cultured rat microglia

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Voltage-dependent potassium channels in activated rat microglia

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Low access resistance perforated patch recordings using amphotericin B.

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T-cell costimulatory molecules B7-1 (CD80) and B7-2 (CD86) are expressed in human microglia but not in astrocytes in culture

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Tumor necrosis factor mediates myelin and oligodendrocyte damage in vitro

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Complement and cytokine gene expression in cultured microglial derived from postmortem human brains

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Platelet-activating factor induced Ca(2+) signaling in human microglia.

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Activation of purinergic P2X receptors inhibits P2Y-mediated Ca2+ influx in human microglia

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DOI

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25 November 2017

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