Differential regulation of phosphatidylserine externalization and DNA fragmentation by caspases in anticancer drug-induced apoptosis of rat mammary adenocarcinoma MTLn3 cells (Q43764281)

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English	Differential regulation of phosphatidylserine externalization and DNA fragmentation by caspases in anticancer drug-induced apoptosis of rat mammary adenocarcinoma MTLn3 cells	scientific article

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scholarly article

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Europe PubMed Central

26 November 2017

Differential regulation of phosphatidylserine externalization and DNA fragmentation by caspases in anticancer drug-induced apoptosis of rat mammary adenocarcinoma MTLn3 cells (English)

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Europe PubMed Central

26 November 2017

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author name string

Huigsloot M

1

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Europe PubMed Central

26 November 2017

Tijdens IB

2

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Europe PubMed Central

26 November 2017

Mulder GJ

3

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Europe PubMed Central

26 November 2017

van de Water B

4

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Europe PubMed Central

26 November 2017

language of work or name

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publication date

1 October 2001

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Europe PubMed Central

26 November 2017

Biochemical Pharmacology

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Europe PubMed Central

26 November 2017

62

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Europe PubMed Central

26 November 2017

8

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Europe PubMed Central

26 November 2017

1087-1097

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Europe PubMed Central

26 November 2017

Apoptosis. Its significance in cancer and cancer therapy

1 reference

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Bcl-2 proteins: regulators of apoptosis or of mitochondrial homeostasis?

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BCL-2 family members and the mitochondria in apoptosis

1 reference

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The inhibitors of apoptosis (IAPs) and their emerging role in cancer

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p53-dependent apoptosis modulates the cytotoxicity of anticancer agents

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The activation of p38 and apoptosis by the inhibition of Erk is antagonized by the phosphoinositide 3-kinase/Akt pathway

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MEKKs, GCKs, MLKs, PAKs, TAKs, and tpls: upstream regulators of the c-Jun amino-terminal kinases?

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Caspases: enemies within

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Death receptors: signaling and modulation

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Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

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Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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Proteolytic activation of protein kinase C delta by an ICE/CED 3-like protease induces characteristics of apoptosis

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Cleavage of focal adhesion kinase by caspases during apoptosis

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Specific cleavage of alpha-fodrin during Fas- and tumor necrosis factor-induced apoptosis is mediated by an interleukin-1beta-converting enzyme/Ced-3 protease distinct from the poly(ADP-ribose) polymerase protease

1 reference

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The role of phosphatidylserine in recognition of apoptotic cells by phagocytes

1 reference

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Early redistribution of plasma membrane phosphatidylserine is a general feature of apoptosis regardless of the initiating stimulus: inhibition by overexpression of Bcl-2 and Abl.

1 reference

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Mechanisms of phosphatidylserine exposure, a phagocyte recognition signal, on apoptotic T lymphocytes

1 reference

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A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD

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Caspase-3 is the primary activator of apoptotic DNA fragmentation via DNA fragmentation factor-45/inhibitor of caspase-activated DNase inactivation.

1 reference

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Caspase-dependent and -independent events in apoptosis induced by hydrogen peroxide

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Protease Involvement in Fodrin Cleavage and Phosphatidylserine Exposure in Apoptosis

1 reference

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Therapeutic efficacy and apoptosis and necrosis kinetics of doxorubicin compared with cisplatin, combined with whole-body hyperthermia in a rat mammary adenocarcinoma

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Dynamics of tumor oxygenation, CD31 staining and transforming growth factor-beta levels after treatment with radiation or cyclophosphamide in the rat 13762 mammary carcinoma

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Fas-mediated apoptosis in mouse hepatocytes involves the processing and activation of caspases

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In situ immunodetection of activated caspase-3 in apoptotic neurons in the developing nervous system

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Apoptosis: cell death defined by caspase activation

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Regulation of phosphatidylserine exposure and phagocytosis of apoptotic T lymphocytes

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Apoptosis in hematopoietic cells (FL5.12) caused by interleukin-3 withdrawal: relationship to caspase activity and the loss of glutathione

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Level of expression of phospholipid scramblase regulates induced movement of phosphatidylserine to the cell surface

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Molecular characterization of mitochondrial apoptosis-inducing factor

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A digitized-fluorescence-imaging study of mitochondrial Ca2+ increase by doxorubicin in cardiac myocytes

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A critical evaluation of the mechanisms of action proposed for the antitumor effects of the anthracycline antibiotics adriamycin and daunorubicin

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Decreased cytotoxic effects of doxorubicin in a human ovarian cancer-cell line expressing wild-type p53 and WAF1/CIP1 genes.

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p53 activates the CD95 (APO-1/Fas) gene in response to DNA damage by anticancer drugs

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CD95 (Fas/APO-1) induces an increased phosphatidylserine synthesis that precedes its externalization during programmed cell death

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Involvement of extracellular calcium in phosphatidylserine exposure during apoptosis

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Interleukin-2-activated natural killer cells can induce both apoptosis and necrosis in rat hepatocytes

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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Annexin V for flow cytometric detection of phosphatidylserine expression on B cells undergoing apoptosis

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7 January 2021

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10.1016/S0006-2952(01)00755-9

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Europe PubMed Central

26 November 2017

1 reference

Europe PubMed Central

26 November 2017

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