p75 Neurotrophin receptor signaling regulates hepatic myofibroblast proliferation and apoptosis in recovery from rodent liver fibrosis. (Q43945393)

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18 January 2020

title

p75 Neurotrophin receptor signaling regulates hepatic myofibroblast proliferation and apoptosis in recovery from rodent liver fibrosis (English)

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18 January 2020

1

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18 January 2020

author name string

Selina Hennedige

2

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18 January 2020

Rebecca L Aucott

3

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18 January 2020

Stephen N Hartland

4

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18 January 2020

Madeleine A Vernon

5

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18 January 2020

R Christopher Benyon

6

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18 January 2020

John P Iredale

7

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18 January 2020

language of work or name

English

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publication date

1 March 2009

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18 January 2020

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18 January 2020

volume

49

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18 January 2020

issue

3

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18 January 2020

page(s)

901-910

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Impact of pegylated interferon alfa-2b and ribavirin on liver fibrosis in patients with chronic hepatitis C.

18 January 2020

cites work

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Activation of hepatic stellate cells--a key issue in liver fibrosis

21 January 2018

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Tissue inhibitor of metalloproteinase-1 messenger RNA expression is enhanced relative to interstitial collagenase messenger RNA in experimental liver injury and fibrosis

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Expression of tissue inhibitor of metalloproteinases 1 and 2 is increased in fibrotic human liver.

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Mechanisms of spontaneous resolution of rat liver fibrosis. Hepatic stellate cell apoptosis and reduced hepatic expression of metalloproteinase inhibitors

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Apoptosis of hepatic stellate cells: involvement in resolution of biliary fibrosis and regulation by soluble growth factors

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Inhibition of inhibitor of kappaB kinases stimulates hepatic stellate cell apoptosis and accelerated recovery from rat liver fibrosis.

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Hepatic stem cells: from inside and outside the liver?

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Neurotrophin signaling through the p75 neurotrophin receptor

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The trk Proto-Oncogene Product: a Signal Transducing Receptor for Nerve Growth Factor

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High-affinity NGF binding requires coexpression of the trk proto-oncogene and the low-affinity NGF receptor

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Differential expression of nerve growth factor receptors leads to altered binding affinity and neurotrophin responsiveness.

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The mature part of proNGF induces the structure of its pro-peptide

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Regulation of cell survival by secreted proneurotrophins

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The nerve growth factor precursor proNGF exhibits neurotrophic activity but is less active than mature nerve growth factor

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Sortilin is essential for proNGF-induced neuronal cell death

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Hepatic stellate cells express the low affinity nerve growth factor receptor p75 and undergo apoptosis in response to nerve growth factor stimulation

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Overexpression of Bcl-2 by activated human hepatic stellate cells: resistance to apoptosis as a mechanism of progressive hepatic fibrogenesis in humans.

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Hepatocytes express nerve growth factor during liver injury: evidence for paracrine regulation of hepatic stellate cell apoptosis

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Human and rat hepatic stellate cells express neurotrophins and neurotrophin receptors

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Spontaneous recovery from micronodular cirrhosis: evidence for incomplete resolution associated with matrix cross-linking

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Gliotoxin stimulates the apoptosis of human and rat hepatic stellate cells and enhances the resolution of liver fibrosis in rats

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Accurate normalization of real-time quantitative RT-PCR data by geometric averaging of multiple internal control genes

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Regulation of alternative macrophage activation by galectin-3.

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'Mature' nerve growth factor is a minor species in most peripheral tissues.

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Molecular forms of NGF in human and rat neuropathic tissues: decreased NGF precursor-like immunoreactivity in human diabetic skin.

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Expression of NGF in hepatocellular carcinoma cells with its receptors in non-tumor cell components.

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Comparison of immunohistochemistry for activated caspase-3 and cleaved cytokeratin 18 with the TUNEL method for quantification of apoptosis in histological sections of PC-3 subcutaneous xenografts

21 January 2018

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The precursor pro-nerve growth factor is the predominant form of nerve growth factor in brain and is increased in Alzheimer's disease

21 January 2018

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Inhibition of apoptosis of activated hepatic stellate cells by tissue inhibitor of metalloproteinase-1 is mediated via effects on matrix metalloproteinase inhibition: implications for reversibility of liver fibrosis

21 January 2018

1 reference

Complete ablation of the neurotrophin receptor p75NTR causes defects both in the nervous and the vascular system

21 January 2018

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Reversibility of hepatic fibrosis in autoimmune hepatitis

21 January 2018

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7 January 2021

Mutation in collagen-1 that confers resistance to the action of collagenase results in failure of recovery from CCl4-induced liver fibrosis, persistence of activated hepatic stellate cells, and diminished hepatocyte regeneration

1 reference

7 January 2021

Regulation of hepatic stellate cell differentiation by the neurotrophin receptor p75NTR

1 reference

7 January 2021

Expression of nerve growth factor (NGF) isoforms in the rat uterus during pregnancy: accumulation of precursor proNGF

1 reference

7 January 2021

Activated hepatic stellate cells overexpress p75NTR after partial hepatectomy and undergo apoptosis on nerve growth factor stimulation

1 reference

7 January 2021

Scar-associated macrophages are a major source of hepatic matrix metalloproteinase-13 and facilitate the resolution of murine hepatic fibrosis

1 reference

7 January 2021

Complete deletion of the neurotrophin receptor p75NTR leads to long-lasting increases in the number of basal forebrain cholinergic neurons.

1 reference

7 January 2021