Metalloprotease inhibitor blocks angiotensin II-induced migration through inhibition of epidermal growth factor receptor transactivation (Q44034950)

29 November 2017

Metalloprotease inhibitor blocks angiotensin II-induced migration through inhibition of epidermal growth factor receptor transactivation (English)

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29 November 2017

Shuichi Saito

1

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29 November 2017

Gerald D Frank

2

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29 November 2017

Evangeline D Motley

3

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29 November 2017

Peter J Dempsey

4

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29 November 2017

Hirotoshi Utsunomiya

5

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29 November 2017

Tadashi Inagami

6

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29 November 2017

Satoru Eguchi

7

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29 November 2017

language of work or name

English

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publication date

1 June 2002

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Biochemical and Biophysical Research Communications

29 November 2017

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29 November 2017

294

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29 November 2017

5

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29 November 2017

1023-1029

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Angiotensin receptors and their antagonists

29 November 2017

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Calcium-dependent epidermal growth factor receptor transactivation mediates the angiotensin II-induced mitogen-activated protein kinase activation in vascular smooth muscle cells

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Epidermal growth factor receptor is indispensable for c-Fos expression and protein synthesis by angiotensin II

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Signal transduction of angiotensin II type 1 receptor through receptor tyrosine kinase

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Angiotensin II type 1 receptor-induced extracellular signal-regulated protein kinase activation is mediated by Ca2+/calmodulin-dependent transactivation of epidermal growth factor receptor.

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N-acetylcysteine inhibits angiotensin ii-mediated activation of extracellular signal-regulated kinase and epidermal growth factor receptor

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Epidermal growth factor receptor transactivation by angiotensin II requires reactive oxygen species in vascular smooth muscle cells.

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Cell communication networks: epidermal growth factor receptor transactivation as the paradigm for interreceptor signal transmission

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EGF receptor transactivation by G-protein-coupled receptors requires metalloproteinase cleavage of proHB-EGF

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Activation of MAPKs by angiotensin II in vascular smooth muscle cells. Metalloprotease-dependent EGF receptor activation is required for activation of ERK and p38 MAPK but not for JNK

7 January 2021

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A metalloprotease-disintegrin, MDC9/meltrin-gamma/ADAM9 and PKCdelta are involved in TPA-induced ectodomain shedding of membrane-anchored heparin-binding EGF-like growth factor.

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Remarkable roles of proteolysis on and beyond the cell surface

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Matrix metalloproteinase-3 releases active heparin-binding EGF-like growth factor by cleavage at a specific juxtamembrane site

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Regulation of vascular smooth muscle cell migration and proliferation in vitro and in injured rat arteries by a synthetic matrix metalloproteinase inhibitor

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Thrombin promotes activation of matrix metalloproteinase-2 produced by cultured vascular smooth muscle cells

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Vascular endothelial growth factor upregulates the expression of matrix metalloproteinases in vascular smooth muscle cells: role of flt-1

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eNOS gene transfer inhibits smooth muscle cell migration and MMP-2 and MMP-9 activity

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Highly selective and orally active inhibitors of type IV collagenase (MMP-9 and MMP-2): N-sulfonylamino acid derivatives

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Identification of an essential signaling cascade for mitogen-activated protein kinase activation by angiotensin II in cultured rat vascular smooth muscle cells. Possible requirement of Gq-mediated p21ras activation coupled to a Ca2+/calmodulin-sensi

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Discovery of CGS 27023A, a non-peptidic, potent, and orally active stromelysin inhibitor that blocks cartilage degradation in rabbits

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Synthesis of a series of stromelysin-selective thiadiazole urea matrix metalloproteinase inhibitors

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Metalloproteinases and their inhibitors in matrix remodeling

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Heparin blockade of thrombin-induced smooth muscle cell migration involves inhibition of epidermal growth factor (EGF) receptor transactivation by heparin-binding EGF-like growth factor

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Metalloprotease-disintegrin MDC9: intracellular maturation and catalytic activity

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Ectodomain shedding of epidermal growth factor receptor ligands is required for keratinocyte migration in cutaneous wound healing

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EGF-genistein inhibits neointimal hyperplasia after vascular injury in an experimental restenosis model

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Central role of the MAPK pathway in ang II-mediated DNA synthesis and migration in rat vascular smooth muscle cells.

7 January 2021

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The G-protein G13 but not G12 mediates signaling from lysophosphatidic acid receptor via epidermal growth factor receptor to Rho.

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Cardiac hypertrophy is inhibited by antagonism of ADAM12 processing of HB-EGF: metalloproteinase inhibitors as a new therapy

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10.1016/S0006-291X(02)00595-8

7 January 2021

Identifiers

DOI

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29 November 2017

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