Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism (Q44068498)

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title

Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism (English)

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6

Stefan Neubauer

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1

Matthias Spindler

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Reinhard Niebler

2

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Helga Remkes

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3

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Michael Horn

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4

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Titus Lanz

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5

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publication date

1 August 2002

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American Journal of Physiology Heart and Circulatory Physiology

29 November 2017

published in

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volume

283

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issue

2

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page(s)

H680-7

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Transport of energy in muscle: the phosphorylcreatine shuttle

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cites work

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Rate equation for creatine kinase predicts the in vivo reaction velocity: 31P NMR surface coil studies in brain, heart, and skeletal muscle of the living rat.

21 January 2018

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Maintained coupling of oxidative phosphorylation to creatine kinase activity in sarcomeric mitochondrial creatine kinase-deficient mice.

21 January 2018

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The cytoplasmic phosphorylation potential. Its possible role in the control of myocardial respiration and cardiac contractility

21 January 2018

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Energetic crosstalk between organelles: architectural integration of energy production and utilization

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

Microassay of free and total creatine from tissue extracts by combination of chromatographic and fluorometric methods

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

Impairment of energy metabolism in intact residual myocardium of rat hearts with chronic myocardial infarction

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

Downregulation of the Na(+)-creatine cotransporter in failing human myocardium and in experimental heart failure

21 January 2018

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Metabolic compartmentation and substrate channelling in muscle cells. Role of coupled creatine kinases in in vivo regulation of cellular respiration--a synthesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

Kinetic, thermodynamic, and developmental consequences of deleting creatine kinase isoenzymes from the heart. Reaction kinetics of the creatine kinase isoenzymes in the intact heart.

21 January 2018

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https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

Diastolic dysfunction and altered energetics in the alphaMHC403/+ mouse model of familial hypertrophic cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

Intracellular compartmentation, structure and function of creatine kinase isoenzymes in tissues with high and fluctuating energy demands: the 'phosphocreatine circuit' for cellular energy homeostasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

Mitochondrial creatine kinase: a key enzyme of aerobic energy metabolism

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00800.2001

10.1152/AJPHEART.00800.2001

21 January 2018

Identifiers

DOI

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29 November 2017

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