Sodium bicarbonate treatment and ubiquitin gene expression in acidotic human subjects with chronic renal failure. (Q44086550)

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11 November 2019

title

Sodium bicarbonate treatment and ubiquitin gene expression in acidotic human subjects with chronic renal failure (English)

1 reference

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human subject research project

11 November 2019

main subject

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author name string

R G Roberts

1

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11 November 2019

K A Graham

3

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11 November 2019

K Bartlett

4

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11 November 2019

R Wilkinson

5

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11 November 2019

publication date

1 July 2002

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European Journal of Clinical Investigation

11 November 2019

published in

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11 November 2019

volume

32

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11 November 2019

issue

7

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11 November 2019

page(s)

488-492

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Skeletal muscle degradation and nitrogen wasting in rats with chronic metabolic acidosis

11 November 2019

cites work

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Metabolic acidosis stimulates protein degradation in rat muscle by a glucocorticoid-dependent mechanism

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Mechanisms of muscle wasting. The role of the ubiquitin-proteasome pathway

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Metabolic acidosis stimulates muscle protein degradation by activating the adenosine triphosphate-dependent pathway involving ubiquitin and proteasomes

21 January 2018

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Isolation of biologically active ribonucleic acid from sources enriched in ribonuclease

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Molecular cloning of cDNA for proteasomes (multicatalytic proteinase complexes) from rat liver: primary structure of the largest component (C2).

21 January 2018

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Protein degradation and increased mRNAs encoding proteins of the ubiquitin-proteasome proteolytic pathway in BC3H1 myocytes require an interaction between glucocorticoids and acidification

21 January 2018

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Differential induction of glucose-regulated and heat shock proteins: effects of pH and sulfhydryl-reducing agents on chicken embryo cells.

21 January 2018

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Cl- current activation in choroid plexus epithelial cells involves a G protein and protein kinase A.

21 January 2018

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The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway

21 January 2018

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Ubiquitin and intracellular protein degradation

21 January 2018

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Muscle wasting in a rat model of long-lasting sepsis results from the activation of lysosomal, Ca2+ -activated, and ubiquitin-proteasome proteolytic pathways.

21 January 2018

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Increased mRNA levels for components of the lysosomal, Ca2+-activated, and ATP-ubiquitin-dependent proteolytic pathways in skeletal muscle from head trauma patients.

21 January 2018

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Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle

21 January 2018

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Sepsis stimulates nonlysosomal, energy-dependent proteolysis and increases ubiquitin mRNA levels in rat skeletal muscle

21 January 2018

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Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription

21 January 2018

1 reference

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10.1046/J.1365-2362.2002.01008.X

21 January 2018

Identifiers

DOI

1 reference

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11 November 2019

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