Effects of caspase inhibition on camptothecin-induced apoptosis of HL-60 cells (Q44124850)

30 November 2017

title

Effects of caspase inhibition on camptothecin-induced apoptosis of HL-60 cells (English)

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30 November 2017

Malcolm A King

1

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30 November 2017

Monica A Radicchi-Mastroianni

series ordinal

2

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30 November 2017

publication date

1 September 2002

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30 November 2017

number of pages

8

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inferred from page(s)

published in

Cytometry

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30 November 2017

volume

49

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30 November 2017

issue

1

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30 November 2017

page(s)

28-35

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Biochemical pathways of caspase activation during apoptosis

30 November 2017

cites work

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https://api.crossref.org/works/10.1002%2FCYTO.10141

Apoptosis induction by caspase-8 is amplified through the mitochondrial release of cytochrome c.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Pro-caspase-3 is a major physiologic target of caspase-8.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Caspase-3 is the primary activator of apoptotic DNA fragmentation via DNA fragmentation factor-45/inhibitor of caspase-activated DNase inactivation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Inhibition of human caspases by peptide-based and macromolecular inhibitors

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Mitochondrial membrane permeabilization during the apoptotic process

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Increased mitochondrial cytochrome c levels and mitochondrial hyperpolarization precede camptothecin-induced apoptosis in Jurkat cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

The S-phase cytotoxicity of camptothecin.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Mitochondria and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Mitochondria as regulators of apoptosis: doubt no more.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Analysis of apoptosis by laser scanning cytometry

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

During apoptosis of HL-60 and U-937 cells caspases are activated independently of dissipation of mitochondrial electrochemical potential

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Collapse of the inner mitochondrial transmembrane potential is not required for apoptosis of HL60 cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Mitochondria and cell death. Mechanistic aspects and methodological issues

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Caspases disrupt mitochondrial membrane barrier function.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Molecular characterization of mitochondrial apoptosis-inducing factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Apoptosis-inducing factor (AIF): a ubiquitous mitochondrial oxidoreductase involved in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Superoxide production by the mitochondrial respiratory chain

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Apoptosis without caspases: an inefficient molecular guillotine?

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

Cell death induced by topoisomerase-targeted drugs: more questions than answers

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

There is substantial nuclear and cellular disintegration before detectable phosphatidylserine exposure during the camptothecin-induced apoptosis of HL-60 cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Mitochondrial control of apoptosis: the role of cytochrome c

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https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Regulation of phosphatidylserine exposure and phagocytosis of apoptotic T lymphocytes

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https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Phagocytosis of nonapoptotic cells dying by caspase-independent mechanisms

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https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Preferential externalization of newly synthesized phosphatidylserine in apoptotic U937 cells is dependent on caspase-mediated pathways

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Flow cytometric detection of apoptosis: Comparison of the assays of in situ DNA degradation and chromatin changes

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

The concentration-dependent diversity of effects of DNA topoisomerase I and II inhibitors on the cell cycle of HL-60 cells*1

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Supravital exposure to propidium iodide identifies apoptotic cells in the absence of nucleosomal DNA fragmentation

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Anti-BrdUrd labeling of newly synthesized DNA in HL-60 cells triggered to apoptosis

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https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

The central executioners of apoptosis: caspases or mitochondria?

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https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Degradation of apoptotic cells and fragments in HL-60 suspension cultures after induction of apoptosis by camptothecin and ethanol

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Superoxide in apoptosis. Mitochondrial generation triggered by cytochrome c loss

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Simultaneous detection of mitochondrial respiratory chain activity and reactive oxygen in digitonin-permeabilized cells using flow cytometry

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Alterations of cell volume regulation in the development of hepatocyte necrosis

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Comparison of methods based on annexin-V binding, DNA content or TUNEL for evaluating cell death in HL-60 and adherent MCF-7 cells

1 reference

https://api.crossref.org/works/10.1002%2FCYTO.10141

7 January 2021

Identifiers

DOI

10.1002/CYTO.10141

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30 November 2017

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