UVB radiation-induced cancer predisposition in Cockayne syndrome group A (Csa) mutant mice. (Q44267626)

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16 December 2019

title

UVB radiation-induced cancer predisposition in Cockayne syndrome group A (Csa) mutant mice (English)

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16 December 2019

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Gijsbertus van der Horst

1

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16 December 2019

Lisiane B Meira

2

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16 December 2019

author name string

Theo G M F Gorgels

3

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16 December 2019

Jan de Wit

4

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16 December 2019

Susana Velasco-Miguel

5

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16 December 2019

James A Richardson

6

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16 December 2019

Yvonne Kamp

7

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16 December 2019

Maaike P G Vreeswijk

8

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16 December 2019

Bep Smit

9

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16 December 2019

Dirk Bootsma

10

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16 December 2019

Jan H J Hoeijmakers

11

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16 December 2019

Errol C Friedberg

12

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16 December 2019

publication date

1 February 2002

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16 December 2019

number of pages

15

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inferred from page(s)

published in

DNA Repair

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16 December 2019

volume

1

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16 December 2019

issue

2

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16 December 2019

page(s)

143-157

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Quality control by DNA repair

16 December 2019

cites work

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The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA.

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Deficient repair of the transcribed strand of active genes in Cockayne's syndrome cells

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ERCC6, a member of a subfamily of putative helicases, is involved in Cockayne's syndrome and preferential repair of active genes

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ATP-dependent chromatin remodeling by the Cockayne syndrome B DNA repair-transcription-coupling factor.

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Recruitment of the putative transcription-repair coupling factor CSB/ERCC6 to RNA polymerase II elongation complexes

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The Cockayne syndrome B protein, involved in transcription-coupled DNA repair, resides in an RNA polymerase II-containing complex

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Interactions involving the human RNA polymerase II transcription/nucleotide excision repair complex TFIIH, the nucleotide excision repair protein XPG, and Cockayne syndrome group B (CSB) protein

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XAB2, a novel tetratricopeptide repeat protein involved in transcription-coupled DNA repair and transcription

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Repair of 8-oxoguanine in DNA is deficient in Cockayne syndrome group B cells

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7 January 2021

Transcription-coupled repair of 8-oxoguanine: requirement for XPG, TFIIH, and CSB and implications for Cockayne syndrome

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7 January 2021

Defective transcription-coupled repair in Cockayne syndrome B mice is associated with skin cancer predisposition

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7 January 2021

Cytogenetic analysis using quantitative, high-sensitivity, fluorescence hybridization

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7 January 2021

High incidence of ultraviolet-B-or chemical-carcinogen-induced skin tumours in mice lacking the xeroderma pigmentosum group A gene

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7 January 2021

Characterization of defective nucleotide excision repair in XPC mutant mice

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7 January 2021

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7 January 2021

The Cockayne syndrome group A gene encodes a WD repeat protein that interacts with CSB protein and a subunit of RNA polymerase II TFIIH

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7 January 2021

p48 Activates a UV-damaged-DNA binding factor and is defective in xeroderma pigmentosum group E cells that lack binding activity

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7 January 2021

Xeroderma pigmentosum p48 gene enhances global genomic repair and suppresses UV-induced mutagenesis

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7 January 2021

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7 January 2021

Molecular cloning and characterization of Saccharomyces cerevisiae RAD28, the yeast homolog of the human Cockayne syndrome A (CSA) gene

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7 January 2021

Increased susceptibility to ultraviolet-B and carcinogens of mice lacking the DNA excision repair gene XPA.

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7 January 2021

Transcription-coupled and global genome repair differentially influence UV-B-induced acute skin effects and systemic immunosuppression

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7 January 2021

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Proneness to UV-induced apoptosis in human fibroblasts defective in transcription coupled repair is associated with the lack of Mdm2 transactivation

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7 January 2021

The relative cytotoxicity of (6-4) photoproducts and cyclobutane dimers in mammalian cells

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7 January 2021

Disruption of the Cockayne syndrome B gene impairs spontaneous tumorigenesis in cancer-predisposed Ink4a/ARF knockout mice

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7 January 2021

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7 January 2021

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Identifiers

DOI

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16 December 2019

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