Functional polymorphisms of circadian positive feedback regulation genes and clinical outcome of Chinese patients with resected colorectal cancer. (Q44342259)

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English	Functional polymorphisms of circadian positive feedback regulation genes and clinical outcome of Chinese patients with resected colorectal cancer.	scientific article

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8 February 2020

Functional polymorphisms of circadian positive feedback regulation genes and clinical outcome of Chinese patients with resected colorectal cancer (English)

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8 February 2020

colorectal cancer

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8 February 2020

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Feng Zhou

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8 February 2020

Xianli He

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8 February 2020

Hanqiang Liu

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8 February 2020

Yong Zhu

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8 February 2020

Tianbo Jin

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8 February 2020

Chao Chen

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8 February 2020

Falin Qu

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8 February 2020

Guoqiang Bao

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8 February 2020

Zhinan Chen

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8 February 2020

Jinliang Xing

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19 July 2011

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8 February 2020

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8 February 2020

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8 February 2020

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8 February 2020

937-946

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8 February 2020

Mammalian circadian biology: elucidating genome-wide levels of temporal organization

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The role of mammalian circadian proteins in normal physiology and genotoxic stress responses.

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Interacting molecular loops in the mammalian circadian clock

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Posttranslational mechanisms regulate the mammalian circadian clock

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BMAL1-dependent circadian oscillation of nuclear CLOCK: posttranslational events induced by dimerization of transcriptional activators of the mammalian clock system

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CLOCK and NPAS2 have overlapping roles in the suprachiasmatic circadian clock

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A CLOCK-less clock

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mCRY1 and mCRY2 are essential components of the negative limb of the circadian clock feedback loop

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Molecular bases for circadian clocks

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Obesity and metabolic syndrome in circadian Clock mutant mice

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Aryl hydrocarbon receptor nuclear translocator-like (BMAL1) is associated with susceptibility to hypertension and type 2 diabetes

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Association between polymorphisms in the Clock gene, obesity and the metabolic syndrome in man.

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Carcinogenicity of shift-work, painting, and fire-fighting

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Ala394Thr polymorphism in the clock gene NPAS2: a circadian modifier for the risk of non-Hodgkin's lymphoma

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Non-synonymous polymorphisms in the circadian gene NPAS2 and breast cancer risk

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Epigenetic inactivation of the circadian clock gene BMAL1 in hematologic malignancies.

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The circadian gene NPAS2 is a novel prognostic biomarker for breast cancer

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Disturbance of circadian gene expression in hepatocellular carcinoma.

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Host circadian clock as a control point in tumor progression.

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Worldwide variations in colorectal cancer

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Cancer statistics, 2010

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Pharmacogenetics: potential for individualized drug therapy through genetics

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Genome-wide interrogation of germline genetic variation associated with treatment response in childhood acute lymphoblastic leukemia

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Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance

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Genetic differences in human circadian clock genes among worldwide populations

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SNPinfo: integrating GWAS and candidate gene information into functional SNP selection for genetic association studies

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Testing the circadian gene hypothesis in prostate cancer: a population-based case-control study

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High-resolution time course analysis of gene expression from pituitary

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Control mechanism of the circadian clock for timing of cell division in vivo

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The circadian gene Period2 plays an important role in tumor suppression and DNA damage response in vivo

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The circadian clock component BMAL1 is a critical regulator of p21WAF1/CIP1 expression and hepatocyte proliferation

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p21 expression in colon cancer and modifying effects of patient age and body mass index on prognosis

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Circadian regulator CLOCK is a histone acetyltransferase

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Cyclin D1 and breast cancer

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CLOCK gene is implicated in weight reduction in obese patients participating in a dietary programme based on the Mediterranean diet

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T3111C clock single nucleotide polymorphism and mood disorders: a meta-analysis

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Genetic variants in human CLOCK associate with total energy intake and cytokine sleep factors in overweight subjects (GOLDN population).

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CLOCK in breast tumorigenesis: genetic, epigenetic, and transcriptional profiling analyses

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c-Myc can induce DNA damage, increase reactive oxygen species, and mitigate p53 function: a mechanism for oncogene-induced genetic instability

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c-myc mRNA overexpression is associated with lymph node metastasis in colorectal cancer

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