Effects of fatty acids in isolated mitochondria: implications for ischemic injury and cardioprotection (Q44470067)

3 December 2017

title

Effects of fatty acids in isolated mitochondria: implications for ischemic injury and cardioprotection (English)

1 reference

3 December 2017

0 references

1 reference

Paavo Korge

1

1 reference

3 December 2017

Henry M Honda

series ordinal

2

1 reference

3 December 2017

James N Weiss

series ordinal

3

1 reference

3 December 2017

publication date

1 July 2003

1 reference

American Journal of Physiology Heart and Circulatory Physiology

3 December 2017

published in

1 reference

3 December 2017

volume

285

1 reference

3 December 2017

issue

1

1 reference

3 December 2017

page(s)

H259-69

1 reference

Lack of oxidative phosphorylation and low mitochondrial membrane potential decrease susceptibility to apoptosis and do not modulate the protective effect of Bcl-x(L) in osteosarcoma cells.

3 December 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Mitochondrial function as a determinant of recovery or death in cell response to injury.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Roles of long chain fatty acids and carnitine in mitochondrial membrane permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Fatty acid import into mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Molecular aspects of the adenine nucleotide carrier from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Protection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Regulation of the mitochondrial permeability transition by matrix Ca(2+) and voltage during anoxia/reoxygenation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

High protonic potential actuates a mechanism of production of reactive oxygen species in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Cardioprotection by ischemic preconditioning preserves mitochondrial function and functional coupling between adenine nucleotide translocase and creatine kinase.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Mitochondrial dysfunction in cardiac disease: ischemia--reperfusion, aging, and heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

The effects of ischaemic preconditioning, diazoxide and 5-hydroxydecanoate on rat heart mitochondrial volume and respiration

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

A novel adenine nucleotide translocase inhibitor, MT-21, induces cytochrome c release by a mitochondrial permeability transition-independent mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Cytochrome c release from mitochondria proceeds by a two-step process

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Opening of mitochondrial K(ATP) channels triggers the preconditioned state by generating free radicals.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Early ischemia-induced alterations of the outer mitochondrial membrane and the intermembrane space: a potential cause for altered energy transfer in cardiac muscle?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Fatty acid-promoted mitochondrial permeability transition by membrane depolarization and binding to the ADP/ATP carrier.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Anion carriers in fatty acid-mediated physiological uncoupling

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

The mechanisms of fatty acid-induced proton permeability of the inner mitochondrial membrane.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

Membrane protein oligomeric structure and transport function

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01028.2002

10.1152/AJPHEART.01028.2002

21 January 2018

Identifiers

DOI

1 reference

3 December 2017

1 reference