1alpha,25(OH)2D3 causes a rapid increase in phosphatidylinositol-specific PLC-beta activity via phospholipase A2-dependent production of lysophospholipid (Q44472909)

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English	1alpha,25(OH)2D3 causes a rapid increase in phosphatidylinositol-specific PLC-beta activity via phospholipase A2-dependent production of lysophospholipid	scientific article

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scholarly article

1 reference

Europe PubMed Central

3 December 2017

1alpha,25(OH)2D3 causes a rapid increase in phosphatidylinositol-specific PLC-beta activity via phospholipase A2-dependent production of lysophospholipid (English)

1 reference

Europe PubMed Central

3 December 2017

5

1 reference

Europe PubMed Central

3 December 2017

author name string

Z Schwartz

1

1 reference

Europe PubMed Central

3 December 2017

D Shaked

2

1 reference

Europe PubMed Central

3 December 2017

R R Hardin

3

1 reference

Europe PubMed Central

3 December 2017

S Gruwell

4

1 reference

Europe PubMed Central

3 December 2017

V L Sylvia

6

1 reference

Europe PubMed Central

3 December 2017

B D Boyan

7

1 reference

Europe PubMed Central

3 December 2017

publication date

1 May 2003

1 reference

Europe PubMed Central

3 December 2017

1 reference

Europe PubMed Central

3 December 2017

68

1 reference

Europe PubMed Central

3 December 2017

5

1 reference

Europe PubMed Central

3 December 2017

423-437

1 reference

Europe PubMed Central

3 December 2017

Differential regulation of growth plate chondrocytes by 1alpha,25-(OH)2D3 and 24R,25-(OH)2D3 involves cell-maturation-specific membrane-receptor-activated phospholipid metabolism

1 reference

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Identification of a membrane receptor for 1,25-dihydroxyvitamin D3 which mediates rapid activation of protein kinase C.

1 reference

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Binding characteristics of a membrane receptor that recognizes 1α25-dihydroxyvitamin D3 and its epimer, 1β,25-dihydroxyvitamin D3

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Maturation-dependent regulation of protein kinase C activity by vitamin D3 metabolites in chondrocyte cultures

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Evidence for involvement of protein kinase C and cyclic adenosine 3',5' monophosphate-dependent protein kinase in the 1,25-dihydroxy-vitamin D3-mediated rapid stimulation of intestinal calcium transport, (transcaltachia).

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Nongenomic activation of the calcium message system by vitamin D metabolites in osteoblast-like cells

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Effect of 1,25(OH)2D3 and 24,25(OH)2D3 on calcium ion fluxes in costochondral chondrocyte cultures

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1,25(OH)2D3 regulates protein kinase C activity through two phospholipid-dependent pathways involving phospholipase A2 and phospholipase C in growth zone chondrocytes

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Evidence for distinct membrane receptors for 1 alpha,25-(OH)(2)D(3) and 24R,25-(OH)(2)D(3) in osteoblasts

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Two alpha subunits of the Gq class of G proteins stimulate phosphoinositide phospholipase C-beta 1 activity

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Involvement of tyrosine kinase activity in 1alpha,25(OH)2-vitamin D3 signal transduction in skeletal muscle cells

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1,25(OH)2-vitamin D-3 stimulates phospholipase A2 activity via a guanine nucleotide-binding protein in chick myoblasts

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Regulation of arachidonic acid turnover by 1,25-(OH)2D3 and 24,25-(OH)2D3 in growth zone and resting zone chondrocyte cultures

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Direct effect of arachidonic acid on protein kinase C and LH-stimulated steroidogenesis in rat Leydig cells; evidence for tonic inhibitory control of steroidogenesis by protein kinase C

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Regulation of prostaglandin E2 production by vitamin D metabolites in growth zone and resting zone chondrocyte cultures is dependent on cell maturation

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Characterization of PGE(2) receptors (EP) and their role as mediators of 1alpha,25-(OH)(2)D(3) effects on growth zone chondrocytes

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Direct effects of 1,25-dihydroxyvitamin D3 and 24,25-dihydroxyvitamin D3 on growth zone and resting zone chondrocyte membrane alkaline phosphatase and phospholipase-A2 specific activities

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1,25-(OH)2D3 and 24,25-(OH)2D3 regulation of arachidonic acid turnover in chondrocyte cultures is cell maturation-specific and may involve direct effects on phospholipase A2.

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Identifiers

10.1016/S0039-128X(03)00044-8

1 reference

Europe PubMed Central

3 December 2017

1 reference

Europe PubMed Central

3 December 2017

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