Dissociation of cytochrome c from the inner mitochondrial membrane during cardiac ischemia (Q44482187)

3 December 2017

Dissociation of cytochrome c from the inner mitochondrial membrane during cardiac ischemia (English)

1 reference

3 December 2017

3

Luke I Szweda

1 reference

3 December 2017

Lech W Czerski

1

1 reference

PubMed ID

12813053

3 December 2017

Pamela A Szweda

series ordinal

2

1 reference

PubMed ID

12813053

3 December 2017

language of work or name

English

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publication date

17 June 2003

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Journal of Biological Chemistry

3 December 2017

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3 December 2017

278

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3 December 2017

36

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3 December 2017

34499-34504

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Development of ischemia-induced damage in defined mitochondrial subpopulations.

3 December 2017

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Interrelationship between cellular calcium homeostasis and free radical generation in myocardial reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Mitochondrial function as a determinant of recovery or death in cell response to injury.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Cardiac reperfusion injury: aging, lipid peroxidation, and mitochondrial dysfunction

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Declines in mitochondrial respiration during cardiac reperfusion: age-dependent inactivation of alpha-ketoglutarate dehydrogenase

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Cytochrome c release occurs via Ca2+-dependent and Ca2+-independent mechanisms that are regulated by Bax.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Cytochrome c release from mitochondria proceeds by a two-step process

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Ionic strength of the intermembrane space of intact mitochondria as estimated with fluorescein-BSA delivered by low pH fusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Complex I-mediated reactive oxygen species generation: modulation by cytochrome c and NAD(P)+ oxidation-reduction state.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Reperfusion injury induces apoptosis in rabbit cardiomyocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Mitochondria as the central control point of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Bcl-2 family: life-or-death switch

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

A mitochondrial perspective on cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

Mitochondrial cytochromes c: a comparative analysis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M302021200

21 January 2018

Identifiers

DOI

10.1074/JBC.M302021200

1 reference

3 December 2017

1 reference

PubMed ID

12813053