Chronic alcohol exposure sensitizes mice to galactosamine-induced liver injury through enhanced keratinocyte chemoattractant and defective IL-10 production. (Q44487649)

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English	Chronic alcohol exposure sensitizes mice to galactosamine-induced liver injury through enhanced keratinocyte chemoattractant and defective IL-10 production.	scientific article

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scholarly article

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5 November 2019

Chronic alcohol exposure sensitizes mice to galactosamine-induced liver injury through enhanced keratinocyte chemoattractant and defective IL-10 production (English)

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5 November 2019

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author name string

Filip Sermon

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Olivier Le Moine

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Eric Quertinmont

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Hubert Louis

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Nathalie Nagy

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Chantal Degraef

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Jacques Devière

8

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1 July 2003

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Journal of Hepatology

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39

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1

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68-76

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5 November 2019

Increased tumor necrosis factor production by monocytes in alcoholic hepatitis

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Excessive in vitro bacterial lipopolysaccharide-induced production of monokines in cirrhosis.

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Increased plasma tumor necrosis factor in severe alcoholic hepatitis

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Increased plasma interleukin-8 concentrations in alcoholic hepatitis

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Intestinal endotoxins as mediators of hepatic injury--an idea whose time has come again

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Exacerbation of alcoholic liver injury by enteral endotoxin in rats.

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Increased intestinal permeability to macromolecules and endotoxemia in patients with chronic alcohol abuse in different stages of alcohol-induced liver disease.

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Antibiotics prevent liver injury in rats following long-term exposure to ethanol

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Inactivation of Kupffer cells prevents early alcohol-induced liver injury

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Galactosamine hepatitis: key role of the nucleotide deficiency period in the pathogenesis of cell injury and cell death

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Galactosamine-induced sensitization to the lethal effects of endotoxin

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Involvement of tumor necrosis factor-alpha in development of hepatic injury in galactosamine-sensitized mice

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ICAM-1 is involved in the mechanism of alcohol-induced liver injury: studies with knockout mice

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NADPH oxidase-derived free radicals are key oxidants in alcohol-induced liver disease

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Toll-like receptor 4 is involved in the mechanism of early alcohol-induced liver injury in mice

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Essential role of tumor necrosis factor alpha in alcohol-induced liver injury in mice

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Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

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Current concepts in the pathogenesis of alcoholic liver injury

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Interleukin-10 and the interleukin-10 receptor

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Cytokine gene expression in cirrhotic and non-cirrhotic human liver

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Activation of nuclear factor kappa B and cytokine imbalance in experimental alcoholic liver disease in the rat

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Interleukin-10 differentially regulates cytokine inhibitor and chemokine release from blood mononuclear cells and fibroblasts

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IL-10 suppresses LPS-induced KC mRNA expression via a translation-dependent decrease in mRNA stability.

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Expression of the chemokine N51/KC in the thymus and epidermis of transgenic mice results in marked infiltration of a single class of inflammatory cells

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Plasma tumor necrosis factor alpha predicts decreased long-term survival in severe alcoholic hepatitis

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The transmembrane form of tumor necrosis factor is the prime activating ligand of the 80 kDa tumor necrosis factor receptor

1 reference

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D-Galactosamine hepatotoxicity is associated with endotoxin sensitivity and mediated by lymphoreticular cells in mice.

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Identifiers

10.1016/S0168-8278(03)00186-7

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5 November 2019

1 reference

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5 November 2019

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