Receptor protein tyrosine phosphatase alpha signaling is involved in androgen depletion-induced neuroendocrine differentiation of androgen-sensitive LNCaP human prostate cancer cells (Q44615404)

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scientific article published in October 2003

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English	Receptor protein tyrosine phosphatase alpha signaling is involved in androgen depletion-induced neuroendocrine differentiation of androgen-sensitive LNCaP human prostate cancer cells	scientific article published in October 2003

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scholarly article

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Europe PubMed Central

4 December 2017

Receptor protein tyrosine phosphatase alpha signaling is involved in androgen depletion-induced neuroendocrine differentiation of androgen-sensitive LNCaP human prostate cancer cells (English)

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Europe PubMed Central

4 December 2017

prostate cancer

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author name string

Xiu-Qing Zhang

1

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Europe PubMed Central

4 December 2017

Dmitry Kondrikov

2

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Europe PubMed Central

4 December 2017

Ta-Chun Yuan

3

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Europe PubMed Central

4 December 2017

Fen-Fen Lin

4

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Europe PubMed Central

4 December 2017

Joel Hansen

5

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4 December 2017

Ming-Fong Lin

6

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4 December 2017

language of work or name

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publication date

1 October 2003

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4 December 2017

number of pages

13

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4 December 2017

22

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4 December 2017

43

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4 December 2017

6704-6716

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Europe PubMed Central

4 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1206764

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Neuroendocrine differentiation in prostatic carcinoma

1 reference

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Neuroendocrine cells in tumour growth of the prostate

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Terminal neuroendocrine differentiation of human prostate carcinoma cells in response to increased intracellular cyclic AMP

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Regulation of distinct stages of skeletal muscle differentiation by mitogen-activated protein kinases

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Differentiation pathways and histogenetic aspects of normal and abnormal prostatic growth: a stem cell model

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Neuro-endocrine cells--a new prognostic parameter in prostate cancer

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Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells

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Activated 3',5'-cyclic AMP-dependent protein kinase is sufficient to induce neuroendocrine-like differentiation of the LNCaP prostate tumor cell line

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Interleukin-6- and cyclic AMP-mediated signaling potentiates neuroendocrine differentiation of LNCaP prostate tumor cells.

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Neuroendocrine differentiation in prostatic malignancy

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Efficient DNA-mediated gene transfer into prostate cancer cell line LNCaP.

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Mitogen-activated protein kinase pathway is involved in the differentiation of muscle cells

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Receptor-like protein-tyrosine phosphatase alpha specifically inhibits insulin-increased prolactin gene expression

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The transmembrane protein tyrosine phosphatase alpha dephosphorylates the insulin receptor in intact cells

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Neuropeptide-induced androgen independence in prostate cancer cells: roles of nonreceptor tyrosine kinases Etk/Bmx, Src, and focal adhesion kinase

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Human prostatic acid phosphatase has phosphotyrosyl protein phosphatase activity

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Decreased expression of cellular prostatic acid phosphatase increases tumorigenicity of human prostate cancer cells

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Tyrosine phosphorylation of c-ErbB-2 is regulated by the cellular form of prostatic acid phosphatase in human prostate cancer cells

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Interaction between protein tyrosine phosphatase and protein tyrosine kinase is involved in androgen-promoted growth of human prostate cancer cells

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Interleukin-6 induces G1 arrest through induction of p27(Kip1), a cyclin-dependent kinase inhibitor, and neuron-like morphology in LNCaP prostate tumor cells

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Etk/Bmx, a tyrosine kinase with a pleckstrin-homology domain, is an effector of phosphatidylinositol 3'-kinase and is involved in interleukin 6-induced neuroendocrine differentiation of prostate cancer cells

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Transfecting well-differentiated prostatic cancer cell line LNCaP.

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Mitogen-activated protein kinases: specific messages from ubiquitous messengers

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10.1038/SJ.ONC.1206764

1 reference

Europe PubMed Central

4 December 2017

1 reference

Europe PubMed Central

4 December 2017

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