Counter-regulation by atorvastatin of gene modulations induced by L-NAME hypertension is associated with vascular protection (Q44628998)

4 December 2017

Counter-regulation by atorvastatin of gene modulations induced by L-NAME hypertension is associated with vascular protection (English)

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Isabelle Brocheriou

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author name string

Morgan Dupuis

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4 December 2017

Isabelle Brocheriou

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Mounsif Haloui

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Liliane Louedec

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Frédérique Capron

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Jean-Baptiste Michel

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Florent Soubrier

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4 December 2017

language of work or name

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5 July 2009

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4 December 2017

Vascular Pharmacology

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51

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253-261

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Determinants of aortic cyclic guanosine monophosphate in hypertension induced by chronic inhibition of nitric oxide synthase

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Atorvastatin limits the pro-inflammatory response of rat aortic smooth muscle cells to thrombin.

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Alteration of flow-induced dilatation in mesenteric resistance arteries of L-NAME treated rats and its partial association with induction of cyclo-oxygenase-2.

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Atorvastatin reduces the expression of cyclooxygenase-2 in a rabbit model of atherosclerosis and in cultured vascular smooth muscle cells.

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Hypertension and prolonged vasoconstrictor signaling in RGS2-deficient mice

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Osteopontin plays an important role in the development of medial thickening and neointimal formation

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Pitavastatin inhibits lysophosphatidic acid-induced proliferation and monocyte chemoattractant protein-1 expression in aortic smooth muscle cells by suppressing Rac-1-mediated reactive oxygen species generation.

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Role of transforming growth factor-beta1 in cardiovascular inflammatory changes induced by chronic inhibition of nitric oxide synthesis

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Statin induction of liver fatty acid-binding protein (L-FABP) gene expression is peroxisome proliferator-activated receptor-alpha-dependent

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Upregulation of endothelial nitric oxide synthase by HMG CoA reductase inhibitors

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Fluvastatin remodels resistance arteries in genetically hypertensive rats, even in the absence of any effect on blood pressure

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Model-based analysis of oligonucleotide arrays: expression index computation and outlier detection

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Phosphatidylinositol-3-kinase signaling mediates vascular smooth muscle cell expression of periostin in vivo and in vitro

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Osteopontin promotes vascular cell adhesion and spreading and is chemotactic for smooth muscle cells in vitro

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Chronic blockade of NO synthase activity induces a proinflammatory phenotype in the arterial wall: prevention by angiotensin II antagonism.

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TGF-β1-induced thrombospondin-1 expression through the p38 MAPK pathway is abolished by fluvastatin in human coronary artery smooth muscle cells

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Improved survival in rats administered NG-nitro L-arginine methyl ester due to converting enzyme inhibition

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Antiinflammatory and antiarteriosclerotic actions of HMG-CoA reductase inhibitors in a rat model of chronic inhibition of nitric oxide synthesis

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Acute antiinflammatory properties of statins involve peroxisome proliferator-activated receptor-alpha via inhibition of the protein kinase C signaling pathway

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Effects of simvastatin on endothelial function after chronic inhibition of nitric oxide synthase by L-NAME.

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Differential regulation of osteoadherin (OSAD) by TGF-beta1 and BMP-2

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Effects of atorvastatin on early recurrent ischemic events in acute coronary syndromes: the MIRACL study: a randomized controlled trial

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Activation of RhoA and inhibition of myosin phosphatase as important components in hypertension in vascular smooth muscle

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Expression of mineralisation-regulating proteins in association with human vascular calcification

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Regulator of G-protein signaling-2 mediates vascular smooth muscle relaxation and blood pressure

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Identification of H-Ras, RhoA, Rac1 and Cdc42 responsive genes.

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Early induction of transforming growth factor-beta via angiotensin II type 1 receptors contributes to cardiac fibrosis induced by long-term blockade of nitric oxide synthesis in rats

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Significance analysis of microarrays applied to the ionizing radiation response

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Statins do more than just lower cholesterol

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Statin monotherapy attenuates renal injury in a salt-sensitive hypertension model of renal disease.

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HMG-CoA reductase inhibitors improve endothelial dysfunction in normocholesterolemic hypertension via reduced production of reactive oxygen species

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Cellular antioxidant effects of atorvastatin in vitro and in vivo

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In vitro differences between venous and arterial-derived smooth muscle cells: potential modulatory role of decorin

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Pitavastatin, an HMG-CoA reductase inhibitor, exerts eNOS-independent protective actions against angiotensin II induced cardiovascular remodeling and renal insufficiency

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Statins activate peroxisome proliferator-activated receptor gamma through extracellular signal-regulated kinase 1/2 and p38 mitogen-activated protein kinase-dependent cyclooxygenase-2 expression in macrophages

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Cyclooxygenase-2 is required for tumor necrosis factor-alpha- and angiotensin II-mediated proliferation of vascular smooth muscle cells

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10.1016/J.VPH.2009.06.011

7 January 2021

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4 December 2017

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