Cholesterol enhances neuron susceptibility to apoptotic stimuli via cAMP/PKA/CREB-dependent up-regulation of Kv2.1. (Q44632335)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:22118516%20AND%20SRC:MED&resulttype=core&format=json

17 January 2020

title

Cholesterol enhances neuron susceptibility to apoptotic stimuli via cAMP/PKA/CREB-dependent up-regulation of Kv2.1. (English)

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17 January 2020

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3

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17 January 2020

Changlong Hu

4

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17 January 2020

author name string

Meng-Hua Zhou

1

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17 January 2020

Guang Yang

2

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17 January 2020

Yan-Ai Mei

5

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17 January 2020

language of work or name

English

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publication date

1 February 2012

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Journal of Neurochemistry

17 January 2020

published in

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17 January 2020

volume

120

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17 January 2020

issue

4

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17 January 2020

page(s)

502-514

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Lipid raft microdomains and neurotransmitter signalling

17 January 2020

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Melatonin receptor agonist 2-iodomelatonin prevents apoptosis of cerebellar granule neurons via K(+) current inhibition

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cAMP/protein kinase A signalling pathway protects against neuronal apoptosis and is associated with modulation of Kv2.1 in cerebellar granule cells

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Activity of adenylyl cyclase and protein kinase A contributes to morphine-induced spinal apoptosis

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Assembly of Trp1 in a signaling complex associated with caveolin-scaffolding lipid raft domains.

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Normotonic cell shrinkage because of disordered volume regulation is an early prerequisite to apoptosis

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Lipid raft disruption protects mature neurons against amyloid oligomer toxicity

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Targeting of ion channels to membrane microdomains: localization of KV channels to lipid rafts.

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Increased membrane cholesterol might render mature hippocampal neurons more susceptible to beta-amyloid-induced calpain activation and tau toxicity

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21 January 2018

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Modulation of endothelial inward-rectifier K+ current by optical isomers of cholesterol

21 January 2018

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