Activation of the 2-5OAS/RNase L pathway in CVB1 or HAV/18f infected FRhK-4 cells does not require induction of OAS1 or OAS2 expression (Q44691585)

5 December 2017

Activation of the 2-5OAS/RNase L pathway in CVB1 or HAV/18f infected FRhK-4 cells does not require induction of OAS1 or OAS2 expression (English)

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5 December 2017

Michael Kulka

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5 December 2017

Mona S Calvo

2

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5 December 2017

Diana T Ngo

3

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5 December 2017

Samantha Q Wales

4

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5 December 2017

Biswendu B Goswami

5

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5 December 2017

language of work or name

English

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publication date

19 April 2009

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5 December 2017

16

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inferred from page(s)

published in

Virology

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5 December 2017

388

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5 December 2017

1

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5 December 2017

169-184

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Cytopathology, plaque assay, and heat inactivation of hepatitis A virus strain HM175.

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Hepatitis A virus inhibits cellular antiviral defense mechanisms induced by double-stranded RNA

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Constitutive expression of (2'-5') oligo A synthetase confers resistance to picornavirus infection.

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Persistent infection of human pancreatic islets by coxsackievirus B is associated with alpha interferon synthesis in beta cells

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RNase L and double-stranded RNA-dependent protein kinase exert complementary roles in islet cell defense during coxsackievirus infection.

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Apoptosis induced by a cytopathic hepatitis A virus is dependent on caspase activation following ribosomal RNA degradation but occurs in the absence of 2'-5' oligoadenylate synthetase

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A phylogenetically conserved RNA structure in the poliovirus open reading frame inhibits the antiviral endoribonuclease RNase L.

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Interferons induce an antiviral state in human pancreatic islet cells

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When two strands are better than one: the mediators and modulators of the cellular responses to double-stranded RNA.

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Gene structure and function of the 2'-5'-oligoadenylate synthetase family.

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Coxsackie B3 virus can replicate in cultured human foetal heart cells and is inhibited by interferon

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The cytopathic 18f strain of Hepatitis A virus induces RNA degradation in FrhK4 cells

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69-kDa and 100-kDa isoforms of interferon-induced (2'-5')oligoadenylate synthetase exhibit differential catalytic parameters.

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The 2-5A system: modulation of viral and cellular processes through acceleration of RNA degradation

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The 100-kDa 2',5'-oligoadenylate synthetase catalyzing preferentially the synthesis of dimeric pppA2'p5'A molecules is composed of three homologous domains

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Characterization of the gene encoding the 100-kDa form of human 2',5' oligoadenylate synthetase.

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20 Interferon-induced enzymes: Microassays and their applications; purification and assay of (2′-5′)-oligoadenylate synthetase and assay of 2′-phosphodiesterase

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Caspase-dependent apoptosis by 2',5'-oligoadenylate activation of RNase L is enhanced by IFN-beta

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Production, purification, and characterization of recombinant 2', 5'-oligoadenylate synthetases

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Novel functions of proteins encoded by viral stress-inducible genes

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RNase L plays a role in the antiviral response to West Nile virus

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Persistent infection of mouse fibroblasts with Coxsackievirus.

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Immunopathology during coxsackievirus infection

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Isoforms p69 and p100 of 2',5'-oligoadenylate synthetase induced differentially by interferons in vivo and in vitro

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Ribosomal RNA cleavage, nuclease activation and 2-5A (ppp(A2′p)nA) in interferon-treated cells

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Some properties of 2-5A binding/nucleolytic activities in gel filtered rabbit reticulocyte lysates

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Identifiers

DOI

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5 December 2017

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