Interaction between trypsinogen isoforms in genetically determined pancreatitis: mutation E79K in cationic trypsin (PRSS1) causes increased transactivation of anionic trypsinogen (PRSS2). (Q44706159)

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scientific article published in January 2004

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English	Interaction between trypsinogen isoforms in genetically determined pancreatitis: mutation E79K in cationic trypsin (PRSS1) causes increased transactivation of anionic trypsinogen (PRSS2).	scientific article published in January 2004

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17 November 2019

Interaction between trypsinogen isoforms in genetically determined pancreatitis: mutation E79K in cationic trypsin (PRSS1) causes increased transactivation of anionic trypsinogen (PRSS2) (English)

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17 November 2019

Miklós Sahin-Tóth

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17 November 2019

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Niels Teich

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Cédric Le Maréchal

2

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Zoltán Kukor

3

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Karel Caca

4

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Helmut Witzigmann

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Jian-Min Chen

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Miklós Tóth

7

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Joachim Mössner

8

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Volker Keim

9

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Claude Férec

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17 November 2019

publication date

1 January 2004

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17 November 2019

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17 November 2019

1

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22-31

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Analysis of progress curves by simulations generated by numerical integration

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Determination of the relative contribution of three genes-the cystic fibrosis transmembrane conductance regulator gene, the cationic trypsinogen gene, and the pancreatic secretory trypsin inhibitor gene-to the etiology of idiopathic chronic pancreat

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Human pancreatic secretory trypsin inhibitor

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Cathepsin B cleavage of the trypsinogen activation peptide

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Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene

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A signal peptide cleavage site mutation in the cationic trypsinogen gene is strongly associated with chronic pancreatitis

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Identifiers

10.1002/HUMU.10285

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17 November 2019

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17 November 2019

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