Sphingomyelin synthase as a potential target for D609-induced apoptosis in U937 human monocytic leukemia cells (Q44707405)

5 December 2017

Sphingomyelin synthase as a potential target for D609-induced apoptosis in U937 human monocytic leukemia cells (English)

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5 December 2017

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6

Yusuf A Hannun

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5 December 2017

Aimin Meng

1

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5 December 2017

Chiara Luberto

2

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5 December 2017

Patrick Meier

3

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5 December 2017

Aiping Bai

4

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5 December 2017

Xiaofeng Yang

5

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5 December 2017

Daohong Zhou

7

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5 December 2017

language of work or name

English

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publication date

1 January 2004

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Experimental Cell Research

5 December 2017

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5 December 2017

292

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2

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5 December 2017

385-392

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Reversion of bovine papillomavirus-induced transformation and immortalization by a xanthate compound

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Selective killing of tumor cells by xanthates

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Induction of apoptosis and potentiation of TNF- and Fas-mediated apoptosis in U937 cells by the xanthogenate compound D609

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Antitumoral activity of a xanthate compound. I. Cytotoxicity studies with neoplastic cell lines in vitro.

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Involvement of SAPK/JNK pathway in X-ray-induced rapid cell death of human T-cell leukemia cell line MOLT-4.

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TNF activates NF-kappa B by phosphatidylcholine-specific phospholipase C-induced "acidic" sphingomyelin breakdown

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Function of the p55 tumor necrosis factor receptor "death domain" mediated by phosphatidylcholine-specific phospholipase C.

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Tumor necrosis factor induces rapid production of 1'2'diacylglycerol by a phosphatidylcholine-specific phospholipase C.

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Interruption of growth signal transduction by an antiviral and antitumoral xanthate compound

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Interruption of TPA-induced signals by an antiviral and antitumoral xanthate compound: inhibition of a phospholipase C-type reaction

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Sphingomyelin synthase, a potential regulator of intracellular levels of ceramide and diacylglycerol during SV40 transformation. Does sphingomyelin synthase account for the putative phosphatidylcholine-specific phospholipase C?

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Differential effects of sphingomyelin hydrolysis and resynthesis on the activation of NF-kappa B in normal and SV40-transformed human fibroblasts

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Basic fibroblast growth factor-induced proliferation of primary astrocytes. evidence for the involvement of sphingomyelin biosynthesis

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Microsomal sphingomyelin accumulation in thioacetamide-injured regenerating rat liver: involvement of sphingomyelin synthase activity

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Reversion of Ras- and phosphatidylcholine-hydrolyzing phospholipase C-mediated transformation of NIH 3T3 cells by a dominant interfering mutant of protein kinase C lambda is accompanied by the loss of constitutive nuclear mitogen-activated protein k

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Ablation of Go alpha-subunit results in a transformed phenotype and constitutively active phosphatidylcholine-specific phospholipase C.

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NIH 3T3 cells stably transfected with the gene encoding phosphatidylcholine-hydrolyzing phospholipase C from Bacillus cereus acquire a transformed phenotype

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Protein kinase C as a drug target: implications for drug or diet prevention and treatment of cancer

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Modulation of the ceramide level, a novel therapeutic concept?

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Enzymes of sphingolipid metabolism: from modular to integrative signaling

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The therapeutic potential of modulating the ceramide/sphingomyelin pathway

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Targeting ceramide metabolism--a strategy for overcoming drug resistance

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Re-examination and further development of a precise and rapid dye method for measuring cell growth/cell kill

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Analysis of apoptotic cells by flow and laser scanning cytometry

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Cross-talk between ceramide and PKC activity in the control of apoptosis in WEHI-231

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Activity of protein kinase C modulates the apoptosis induced by polychlorinated biphenyls in human leukemic HL-60 cells

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Tumor necrosis factor induces necrosis of human carcinoma xenografts in the presence of tricyclodecan-9-yl-xanthogenate and lauric acid

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Daunorubicin- and mitoxantrone-triggered phosphatidylcholine hydrolysis: implication in drug-induced ceramide generation and apoptosis

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Modulation of protein kinase C in antitumor treatment

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7 January 2021

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Identifiers

DOI

10.1016/J.YEXCR.2003.10.001

1 reference

5 December 2017

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