Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP. (Q44748745)

5 December 2017

title

Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP. (English)

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5 December 2017

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Daming Zhuang

1

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5 December 2017

Alice-Corina Ceacareanu

2

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5 December 2017

Yi Lin

3

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5 December 2017

Bogdan Ceacareanu

4

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5 December 2017

Madhulika Dixit

5

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5 December 2017

Kenneth E Chapman

6

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5 December 2017

Christopher M Waters

7

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5 December 2017

Gadiparthi N Rao

8

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5 December 2017

Aviv Hassid

9

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5 December 2017

publication date

29 January 2004

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American Journal of Physiology Heart and Circulatory Physiology

5 December 2017

published in

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5 December 2017

volume

286

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5 December 2017

issue

6

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5 December 2017

page(s)

H2103-12

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Overexpression of human catalase inhibits proliferation and promotes apoptosis in vascular smooth muscle cells

5 December 2017

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Regulation of insulin-like growth factor type I (IGF-I) receptor kinase activity by protein tyrosine phosphatase 1B (PTP-1B) and enhanced IGF-I-mediated suppression of apoptosis and motility in PTP-1B-deficient fibroblasts

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Nitric oxide-induced motility in aortic smooth muscle cells: role of protein tyrosine phosphatase SHP-2 and GTP-binding protein Rho.

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Diabetes accelerates smooth muscle accumulation in lesions of atherosclerosis: lack of direct growth-promoting effects of high glucose levels

21 January 2018

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21 January 2018

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Targeted overexpression of IGF-I in smooth muscle cells of transgenic mice enhances neointimal formation through increased proliferation and cell migration after intraarterial injury

21 January 2018

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21 January 2018

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10.1152/AJPHEART.01118.2003

21 January 2018

Identifiers

DOI

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5 December 2017

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