Beta-amyloid peptide at sublethal concentrations downregulates brain-derived neurotrophic factor functions in cultured cortical neurons. (Q44752355)

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31 December 2019

title

Beta-amyloid peptide at sublethal concentrations downregulates brain-derived neurotrophic factor functions in cultured cortical neurons (English)

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31 December 2019

author name string

Liqi Tong

1

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31 December 2019

Robert Balazs

2

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31 December 2019

Phillip L Thornton

3

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31 December 2019

Carl W Cotman

4

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31 December 2019

language of work or name

English

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publication date

1 July 2004

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31 December 2019

published in

Journal of Neuroscience

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31 December 2019

volume

24

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31 December 2019

issue

30

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31 December 2019

page(s)

6799-6809

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Plaque-independent disruption of neural circuits in Alzheimer's disease mouse models

31 December 2019

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Brain-derived neurotrophic factor triggers a rapid glutamate release through increase of intracellular Ca(2+) and Na(+) in cultured cerebellar neurons

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Hippocampal long-term potentiation is impaired in mice lacking brain-derived neurotrophic factor

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Clusterin (apoJ) Alters the Aggregation of Amyloid β-Peptide (Aβ1-42) and Forms Slowly Sedimenting Aβ Complexes That Cause Oxidative Stress

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TrkB and TrkC signaling are required for maturation and synaptogenesis of hippocampal connections.

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10.1523/JNEUROSCI.5463-03.2004

Identifiers

DOI

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31 December 2019

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31 December 2019

PubMed publication ID

15282285

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