The coxsackievirus 2B protein suppresses apoptotic host cell responses by manipulating intracellular Ca2+ homeostasis. (Q44771677)

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24 November 2019

title

The coxsackievirus 2B protein suppresses apoptotic host cell responses by manipulating intracellular Ca2+ homeostasis (English)

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24 November 2019

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Rosario Rizzuto

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Willem Melchers

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Peter H Willems

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Frank J. M. van Kuppeveld

24 November 2019

8

object named as

Frank J M van Kuppeveld

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3

Kjerstin W H Lanke

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Michelangelo Campanella

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object named as

Michelangelo Campanella

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author name string

Arjan S de Jong

2

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language of work or name

English

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publication date

19 February 2004

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Journal of Biological Chemistry

24 November 2019

published in

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volume

279

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issue

18

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page(s)

18440-18450

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A time to kill: viral manipulation of the cell death program

24 November 2019

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Coxsackievirus protein 2B modifies endoplasmic reticulum membrane and plasma membrane permeability and facilitates virus release.

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Cellular COPII proteins are involved in production of the vesicles that form the poliovirus replication complex.

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Multimerization reactions of coxsackievirus proteins 2B, 2C and 2BC: a mammalian two-hybrid analysis.

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Viroporin-mediated membrane permeabilization. Pore formation by nonstructural poliovirus 2B protein.

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Homomultimerization of the coxsackievirus 2B protein in living cells visualized by fluorescence resonance energy transfer microscopy

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Chimeric green fluorescent protein as a tool for visualizing subcellular organelles in living cells

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Rapid changes of mitochondrial Ca2+ revealed by specifically targeted recombinant aequorin

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Transfected aequorin in the measurement of cytosolic Ca2+ concentration ([Ca2+]c). A critical evaluation.

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The Golgi apparatus is an inositol 1,4,5-trisphosphate-sensitive Ca2+ store, with functional properties distinct from those of the endoplasmic reticulum

21 January 2018

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Dynamics of [Ca2+] in the endoplasmic reticulum and cytoplasm of intact HeLa cells. A comparative study

21 January 2018

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Close contacts with the endoplasmic reticulum as determinants of mitochondrial Ca2+ responses

21 January 2018

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Reduced loading of intracellular Ca(2+) stores and downregulation of capacitative Ca(2+) influx in Bcl-2-overexpressing cells.

21 January 2018

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Recombinant expression of the voltage-dependent anion channel enhances the transfer of Ca2+ microdomains to mitochondria

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The Ca2+ concentration of the endoplasmic reticulum is a key determinant of ceramide-induced apoptosis: significance for the molecular mechanism of Bcl-2 action

21 January 2018

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Structure-function analysis of coxsackie B3 virus protein 2B.

21 January 2018

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Bcl-2 targeted to the endoplasmic reticulum can inhibit apoptosis induced by Myc but not etoposide in Rat-1 fibroblasts

21 January 2018

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Endoplasmic reticulum localized Bcl-2 prevents apoptosis when redistribution of cytochrome c is a late event

21 January 2018

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BCL-2 family members and the mitochondria in apoptosis

21 January 2018

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Regulation of mitochondrial ATP synthesis by calcium: evidence for a long-term metabolic priming

21 January 2018

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Bcl-2 decreases the free Ca2+ concentration within the endoplasmic reticulum

21 January 2018

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21 January 2018

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Direct cleavage by the calcium-activated protease calpain can lead to inactivation of caspases

21 January 2018

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21 January 2018

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Bcl-2 and Bcl-xL overexpression inhibits cytochrome c release, activation of multiple caspases, and virus release following coxsackievirus B3 infection.

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21 January 2018

Identifiers

DOI

10.1074/JBC.M309494200

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14976205%20AND%20SRC:MED&resulttype=core&format=json

24 November 2019

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