Long-lasting decrease in neuronal Ca2+/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent seizures (Q44867664)

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scientific article published in December 1999

Language	Label	Description	Also known as
English	Long-lasting decrease in neuronal Ca2+/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent seizures	scientific article published in December 1999

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scholarly article

1 reference

Europe PubMed Central

7 December 2017

Long-lasting decrease in neuronal Ca2+/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent seizures (English)

1 reference

Europe PubMed Central

7 December 2017

author name string

Blair RE

1

1 reference

Europe PubMed Central

7 December 2017

Churn SB

2

1 reference

Europe PubMed Central

7 December 2017

Sombati S

3

1 reference

Europe PubMed Central

7 December 2017

Lou JK

4

1 reference

Europe PubMed Central

7 December 2017

DeLorenzo RJ

5

1 reference

Europe PubMed Central

7 December 2017

language of work or name

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publication date

1 December 1999

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Europe PubMed Central

7 December 2017

number of pages

12

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based on heuristic

inferred from page(s)

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Europe PubMed Central

7 December 2017

851

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Europe PubMed Central

7 December 2017

1-2

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Europe PubMed Central

7 December 2017

54-65

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Europe PubMed Central

7 December 2017

Effects of intracellular calcium on GABAA receptors in mouse cortical neurons

1 reference

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Regulation of gene expression in hippocampal neurons by distinct calcium signaling pathways

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Further observations on hippocampal neurons in dispersed cell culture

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Regulation of type-II calmodulin kinase: functional implications

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Zinc-induced collapse of augmented inhibition by GABA in a temporal lobe epilepsy model.

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Limbic epilepsy in transgenic mice carrying a Ca2+/calmodulin-dependent kinase II alpha-subunit mutation

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Okadaic acid, a protein phosphatase inhibitor, inhibits nerve growth factor-directed neurite outgrowth in PC12 cells

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Exposure of hippocampal slices to magnesium-free medium produces epileptiform activity and simultaneously decreases calcium and calmodulin-dependent protein kinase II activity

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Modulation of GABAergic receptor binding by activation of calcium and calmodulin-dependent kinase II membrane phosphorylation

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Excitotoxic activation of the NMDA receptor results in inhibition of calcium/calmodulin kinase II activity in cultured hippocampal neurons.

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Excitotoxicity affects membrane potential and calmodulin kinase II activity in cultured rat cortical neurons

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Global forebrain ischemia induces a posttranslational modification of multifunctional calcium- and calmodulin-dependent kinase II

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Global forebrain ischemia results in decreased immunoreactivity of calcium/calmodulin-dependent protein kinase II

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Electrophysiology of glutamate neurotoxicity in vitro: induction of a calcium-dependent extended neuronal depolarization

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The calmodulin hypothesis of neurotransmission

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Prolonged activation of the N-methyl-D-aspartate receptor-Ca2+ transduction pathway causes spontaneous recurrent epileptiform discharges in hippocampal neurons in culture

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Regional distribution of type II Ca2+/calmodulin-dependent protein kinase in rat brain

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Calcium signaling in neurons: molecular mechanisms and cellular consequences

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Physiological and pharmacological alterations in postsynaptic GABA(A) receptor function in a hippocampal culture model of chronic spontaneous seizures

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Association of calmodulin-dependent kinase II and its substrate proteins with neuronal cytoskeleton.

1 reference

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Kindling induces a long-lasting change in the activity of a hippocampal membrane calmodulin-dependent protein kinase system.

1 reference

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Expression of a multifunctional Ca2+/calmodulin-dependent protein kinase and mutational analysis of its autoregulation

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Neuronal Ca2+/calmodulin-dependent protein kinases

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Calcium . calmodulin-dependent protein kinase II and calcium . phospholipid-dependent protein kinase activities in rat tissues assayed with a synthetic peptide

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An improved method to determine cell viability by simultaneous staining with fluorescein diacetate-propidium iodide

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Hippocampal kindling leads to different changes in paired-pulse depression of local evoked field potentials in CA1 area and in fascia dentata

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Loss of inhibition precedes delayed spontaneous seizures in the hippocampus after tetanic electrical stimulation

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Loss of GABAA receptors during partial status epilepticus

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Rapid seizure-induced reduction of benzodiazepine and Zn2+ sensitivity of hippocampal dentate granule cell GABAA receptors

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Intracellular calcium mobilization and neurite outgrowth in mammalian neurons

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Calcineurin is a common target of cyclophilin-cyclosporin A and FKBP-FK506 complexes

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Functional anatomy of hippocampal seizures

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Intracellular injections of EGTA block induction of hippocampal long-term potentiation.

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Antiepileptic drug mechanisms of action

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Postsynaptic calcium is sufficient for potentiation of hippocampal synaptic transmission.

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Phosphorylation and regulation of glutamate receptors by calcium/calmodulin-dependent protein kinase II.

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Update on the mechanism of action of antiepileptic drugs

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Calcium activates serum response factor-dependent transcription by a Ras- and Elk-1-independent mechanism that involves a Ca2+/calmodulin-dependent kinase

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Loss of type II calcium/calmodulin-dependent kinase activity correlates with stages of development of electrographic seizures in status epilepticus in rat

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Neurotoxic activation of glutamate receptors induces an extended neuronal depolarization in cultured hippocampal neurons

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Recurrent spontaneous seizure activity in hippocampal neuronal networks in culture

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Alpha-subunit of calcium/calmodulin-dependent protein kinase II enhances gamma-aminobutyric acid and inhibitory synaptic responses of rat neurons in vitro

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Kindling alters the calcium/calmodulin-dependent phosphorylation of synaptic plasma membrane proteins in rat hippocampus

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Identifiers

10.1016/S0006-8993(99)02100-9

1 reference

Europe PubMed Central

7 December 2017

1 reference

Europe PubMed Central

7 December 2017

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