Brain lactate uptake increases at the site of impact after traumatic brain injury (Q44870598)

7 December 2017

Brain lactate uptake increases at the site of impact after traumatic brain injury (English)

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7 December 2017

traumatic brain injury

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author name string

Chen T

1

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7 December 2017

Qian YZ

2

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7 December 2017

Rice A

3

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7 December 2017

Zhu JP

4

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7 December 2017

Di X

5

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7 December 2017

Bullock R

6

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7 December 2017

language of work or name

English

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publication date

1 April 2000

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7 December 2017

7

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based on heuristic

inferred from page(s)

published in

Brain Research

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7 December 2017

861

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7 December 2017

2

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7 December 2017

281-287

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Lactate transport through the blood-retinal and the blood-brain barrier in rats.

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Effect of posttraumatic hypoventilation on cerebral energy metabolism

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Functional compartmentalization of energy production in neural tissue

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Post-traumatic selective stimulation of glycolysis

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Clinical significance of CSF glutamate concentrations following severe traumatic brain injury in humans.

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Evidence for prolonged release of excitatory amino acids in severe human head trauma. Relationship to clinical events

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Factors affecting excitatory amino acid release following severe human head injury

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Lactate/glucose dynamics after rat fluid percussion brain injury

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Kinetics of blood-brain barrier transport of pyruvate, lactate and glucose in suckling, weanling and adult rats

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Inhibition, by 2-oxo acids that accumulate in maple-syrup-urine disease, of lactate, pyruvate, and 3-hydroxybutyrate transport across the blood-brain barrier.

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Serial CSF lactate-pyruvate values as a guide to prognosis in head injury coma.

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Voltage-dependent Na+/K+ ion channel blockade fails to ameliorate behavioral deficits after traumatic brain injury in the rat.

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A fluid percussion model of experimental brain injury in the rat

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Metabolic alterations in brain during anoxic-anoxia and subsequent recovery.

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Lactate as energy source for brain in glucose-6-phosphatase deficient child

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The blood-brain barrier disruption to circulating proteins in the early period after fluid percussion brain injury in rats

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Induction processes in blood-brain transfer of ketone bodies during starvation

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The neuronal environment after subarachnoid haemorrhage--correlation of amino acid and nucleoside levels with post-operative recovery

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Brain energy metabolism and function during hepatic encephalopathy.

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Regional transport of some essential nutrients across the blood-brain barrier in normal and diseased states

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Reversibility of ischemic brain damage.

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Production and clearance of lactate from brain tissue, cerebrospinal fluid, and serum following experimental brain injury

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Lactate accumulation following concussive brain injury: the role of ionic fluxes induced by excitatory amino acids.

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Acid-base balance and arterial and CSF lactate levels following human head injury

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Effects of mild hypothermia on cerebral blood flow-independent changes in cortical extracellular levels of amino acids following contusion trauma in the rat.

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Autoradiographic measurement of cerebral lactate transport rate constants in normal and activated conditions

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Lactate metabolism and its effects on glucose metabolism in an excised neural tissue

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Influence of tissue acidosis upon restitution of brain energy metabolism following total ischemia

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Cerebral blood flow and metabolic rate of oxygen, glucose, lactate, pyruvate, ketone bodies and amino acids

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Traumatic brain injury in the rat: characterization of a lateral fluid-percussion model.

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Neurotransmitters regulate energy metabolism in astrocytes: implications for the metabolic trafficking between neural cells.

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Blood Brain Barrier Permeability to Lactate

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pH dependence of blood-brain barrier permeability to lactate and nicotine

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Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization

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Excitatory amino acids stimulate aerobic glycolysis in astrocytes via an activation of the Na+/K+ ATPase

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Vascular permeability alterations to horseradish peroxidase in experimental brain injury

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Brain Lactic Acidosis and Ischemic Cell Damage: 1. Biochemistry and Neurophysiology

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Cerebral energy reserves after prolonged hypoxia and ischemia.

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Cell damage in the brain: a speculative synthesis

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Lactate-Supported Synaptic Function in the Rat Hippocampal Slice Preparation

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Brain lactate, not glucose, fuels the recovery of synaptic function from hypoxia upon reoxygenation: an in vitro study

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Increase in extracellular glutamate caused by reduced cerebral perfusion pressure and seizures after human traumatic brain injury: a microdialysis study.

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Continuous monitoring of cerebral substrate delivery and clearance: initial experience in 24 patients with severe acute brain injuries

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Partitioning of CO2 production between glucose and lactate in excised sympathetic ganglia, with implications for brain.

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inferred from DOI database lookup

7 January 2021

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Identifiers

DOI

10.1016/S0006-8993(00)01992-2

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7 December 2017

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