Histone deacetylase 4 selectively contributes to podocyte injury in diabetic nephropathy (Q44996558)

8 December 2017

Histone deacetylase 4 selectively contributes to podocyte injury in diabetic nephropathy (English)

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8 December 2017

Xiaojie Wang

1

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8 December 2017

Jiang Liu

2

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8 December 2017

Junhui Zhen

3

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8 December 2017

Chun Zhang

4

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8 December 2017

Qiang Wan

5

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8 December 2017

Guangyi Liu

6

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8 December 2017

Xinbing Wei

7

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8 December 2017

Yan Zhang

8

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8 December 2017

Ziying Wang

9

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8 December 2017

Huirong Han

10

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8 December 2017

Huiyan Xu

11

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8 December 2017

Chanchan Bao

12

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8 December 2017

Zhenyu Song

13

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8 December 2017

Xiumei Zhang

14

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8 December 2017

Ningjun Li

15

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8 December 2017

Fan Yi

16

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8 December 2017

language of work or name

English

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publication date

9 April 2014

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8 December 2017

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8 December 2017

86

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8 December 2017

712-725

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8 December 2017

4

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https://scigraph.springernature.com/pub.10.1038/ki.2014.111

8 December 2017

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Histone deacetylase-2 is a key regulator of diabetes- and transforming growth factor-beta1-induced renal injury

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Long-term administration of the histone deacetylase inhibitor vorinostat attenuates renal injury in experimental diabetes through an endothelial nitric oxide synthase-dependent mechanism

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7 January 2021

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7 January 2021

Autophagy attenuates diabetic glomerular damage through protection of hyperglycemia-induced podocyte injury

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7 January 2021

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Autophagy plays an essential role in the clearance of Pseudomonas aeruginosa by alveolar macrophages

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7 January 2021

The podocyte and diabetes mellitus: is the podocyte the key to the origins of diabetic nephropathy?

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7 January 2021

Podocyte injury and glomerulosclerosis in hyperhomocysteinemic rats

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice

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7 January 2021

Vps34 deficiency reveals the importance of endocytosis for podocyte homeostasis

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HDAC4-regulated STAT1 activation mediates platinum resistance in ovarian cancer.

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Loss of TIMP3 underlies diabetic nephropathy via FoxO1/STAT1 interplay

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7 January 2021

Suppressors of cytokine signaling abrogate diabetic nephropathy

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7 January 2021

Enhanced expression of Janus kinase-signal transducer and activator of transcription pathway members in human diabetic nephropathy

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7 January 2021

Cytokines tumor necrosis factor-α and interferon-γ induce pancreatic β-cell apoptosis through STAT1-mediated Bim protein activation

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7 January 2021

Short interfering RNA against STAT1 attenuates cisplatin-induced ototoxicity in the rat by suppressing inflammation

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7 January 2021

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7 January 2021

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7 January 2021

The protective role of Smad7 in diabetic kidney disease: mechanism and therapeutic potential

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7 January 2021

Contribution of guanine nucleotide exchange factor Vav2 to hyperhomocysteinemic glomerulosclerosis in rats

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7 January 2021

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7 January 2021

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7 January 2021

Kidney-specific reconstitution of the A1 adenosine receptor in A1 adenosine receptor knockout mice reduces renal ischemia-reperfusion injury

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7 January 2021

NKCC2A and NFAT5 regulate renal TNF production induced by hypertonic NaCl intake

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7 January 2021

Expression and regulation of a novel identified TNFAIP8 family is associated with diabetic nephropathy

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7 January 2021

TGF-β induces acetylation of chromatin and of Ets-1 to alleviate repression of miR-192 in diabetic nephropathy

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7 January 2021

NOD2 promotes renal injury by exacerbating inflammation and podocyte insulin resistance in diabetic nephropathy

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7 January 2021

Podocytes in culture: past, present, and future

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7 January 2021

Formation of lipid raft redox signalling platforms in glomerular endothelial cells: an early event of homocysteine-induced glomerular injury

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7 January 2021

Novel role of NOD2 in mediating Ca2+ signaling: evidence from NOD2-regulated podocyte TRPC6 channels in hyperhomocysteinemia

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7 January 2021

Identifiers

DOI

10.1038/KI.2014.111

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8 December 2017

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