Multiple pathological events in exercised dystrophic mdx mice are targeted by pentoxifylline: outcome of a large array of in vivo and ex vivo tests. (Q45129880)

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scientific article published on 8 January 2009

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English	Multiple pathological events in exercised dystrophic mdx mice are targeted by pentoxifylline: outcome of a large array of in vivo and ex vivo tests.	scientific article published on 8 January 2009

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19 January 2020

Multiple pathological events in exercised dystrophic mdx mice are targeted by pentoxifylline: outcome of a large array of in vivo and ex vivo tests (English)

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19 January 2020

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Antonella Liantonio

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19 January 2020

Annamaria De Luca

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19 January 2020

Jean-Francois Rolland

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19 January 2020

Beniamino Palmieri

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19 January 2020

monica montagnani

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19 January 2020

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Rosa Burdi

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19 January 2020

Bodvael Fraysse

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19 January 2020

Karina Litvinova

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19 January 2020

Anna Cozzoli

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19 January 2020

Viviana Giannuzzi

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19 January 2020

Giulia Maria Camerino

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Valeriana Sblendorio

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19 January 2020

Roberta Francesca Capogrosso

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19 January 2020

Francesca Andreetta

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19 January 2020

Paolo Confalonieri

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19 January 2020

Leonarda De Benedictis

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19 January 2020

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8 January 2009

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19 January 2020

Journal of Applied Physiology

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19 January 2020

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19 January 2020

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19 January 2020

1311-1324

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19 January 2020

How calcium influx through calcium leak channels is responsible for the elevated levels of calcium-dependent proteolysis in dystrophic myotubes

1 reference

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Oxidative stress as a therapeutic target during muscle wasting: considering the complex interactions.

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Primary role of functional ischemia, quantitative evidence for the two-hit mechanism, and phosphodiesterase-5 inhibitor therapy in mouse muscular dystrophy

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Bcl-2 overexpression prevents calcium overload and subsequent apoptosis in dystrophic myotubes

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Phosphodiesterase inhibitors.

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Endurance capacity in maturing mdx mice is markedly enhanced by combined voluntary wheel running and green tea extract

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Protein kinase A signalling via CREB controls myogenesis induced by Wnt proteins

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A multidisciplinary evaluation of the effectiveness of cyclosporine a in dystrophic mdx mice.

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Gentamicin treatment in exercised mdx mice: Identification of dystrophin-sensitive pathways and evaluation of efficacy in work-loaded dystrophic muscle

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21 January 2018

Enhanced dystrophic progression in mdx mice by exercise and beneficial effects of taurine and insulin-like growth factor-1.

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21 January 2018

Green tea extract and its major polyphenol (-)-epigallocatechin gallate improve muscle function in a mouse model for Duchenne muscular dystrophy.

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21 January 2018

Sarcolemmal damage in dystrophin deficiency is modulated by synergistic interactions between mechanical and oxidative/nitrosative stresses

1 reference

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21 January 2018

The alteration of calcium homeostasis in adult dystrophic mdx muscle fibers is worsened by a chronic exercise in vivo

1 reference

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21 January 2018

Pentoxifylline fails to attenuate fibrosis in dystrophic (mdx) diaphragm muscle

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Anti-TNFalpha (Remicade) therapy protects dystrophic skeletal muscle from necrosis.

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Sildenafil and cardiomyocyte-specific cGMP signaling prevent cardiomyopathic changes associated with dystrophin deficiency

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Regulation of Nox1 activity via protein kinase A-mediated phosphorylation of NoxA1 and 14-3-3 binding.

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Mechanical stress activates the nuclear factor-kappaB pathway in skeletal muscle fibers: a possible role in Duchenne muscular dystrophy.

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Pentoxifylline attenuates tubulointerstitial fibrosis by blocking Smad3/4-activated transcription and profibrogenic effects of connective tissue growth factor.

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Effect of torbafylline on mitochondrial calmitine in mouse skeletal muscle regeneration after injection of a myotoxic drug

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Force and power output of diaphragm muscle strips from mdx and control mice after clenbuterol treatment

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Lipid peroxidation inhibition blunts nuclear factor-kappaB activation, reduces skeletal muscle degeneration, and enhances muscle function in mdx mice.

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Endothelial dysfunction in mice with streptozotocin-induced type 1 diabetes is opposed by compensatory overexpression of cyclooxygenase-2 in the vasculature

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Follistatin induction by nitric oxide through cyclic GMP: a tightly regulated signaling pathway that controls myoblast fusion

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Oxidative stress and the pathogenesis of muscular dystrophies

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Overactivity of exercise-sensitive cation channels and their impaired modulation by IGF-1 in mdx native muscle fibers: beneficial effect of pentoxifylline.

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Xanthine derivatives as antagonists at A1 and A2 adenosine receptors

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Overexpression of a calpastatin transgene in mdx muscle reduces dystrophic pathology

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Helper (CD4(+)) and cytotoxic (CD8(+)) T cells promote the pathology of dystrophin-deficient muscle

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Involvement of TRPC in the abnormal calcium influx observed in dystrophic (mdx) mouse skeletal muscle fibers

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